Traits - Food Intake Control

Behavioral Response

Food Intake Control

Food intake control refers to the biological systems that signal hunger, fullness, and food reward, balancing energy intake against energy needs.

GWAS work has identified variants near hypothalamic and reward-circuit genes (such as FTO, MC4R, and BDNF) linked to differences in appetite and intake control.

This result reflects research-level variation in appetite biology and is not a measure of self-discipline or a diet plan.

Sleep, stress, food environment, meal composition, hormones, medications, and culture all strongly shape day-to-day eating.

12 SNPs

1 Study

Oct 14, 2026

DEMO REPORT

Your Analysis

Genetic Tendency

Based on your genetic markers, your tendency toward this trait is shown below

Your Result

Slightly higher food addiction

Very Low Very High

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Confidence Score

Very high

This score indicates how strongly your genetics predispose you to this trait based on 12/12 SNPs detected.

Trait Influence Breakdown

Genetic

65%

Environment

35%

Scientific Evidence

Understanding the Data

SNP: A specific genetic marker relevant to this trait (e.g., rs2588978)
Genotype: Your genetic makeup at the given SNP location (e.g., CC)
Variant allele: The alternative DNA sequence at the SNP site

Variant allele frequency: Percentage of population carrying this variant
Variant found: Whether the variant was detected in your DNA file

Variant Detected

FTO GeneCards

This gene is a nuclear protein of the AlkB related non-haem iron and 2-oxoglutarate-dependent oxygenase superfamily but the exact physiological function of this gene is not known. Other non-heme iron enzymes function to reverse alkylated DNA and RNA damage by oxidative demethylation. Studies in mice and humans indicate a role in nervous and cardiovascular systems and a strong association with body mass index, obesity risk, and type 2 diabetes. [provided by RefSeq, Jul 2011]

Genomic Location

Chr 16 Start: 53,701,692 End: 54,158,512 Build: HG19

5' end 3' end

Associated SNPs

1 / 1 detected

SNP	Genotype	Ref. Allele	Variant Allele	Frequency	Status
rs9939609	A T	T	A	34.01%	Detected
Your Genotype AT Reference Allele T Variant Allele A Population Frequency 34.01% Variant Status Variant Detected Database Link NCBI dbSNP

Variant Detected

LEPR GeneCards

The protein encoded by this gene belongs to the gp130 family of cytokine receptors that are known to stimulate gene transcription via activation of cytosolic STAT proteins. This protein is a receptor for leptin (an adipocyte-specific hormone that regulates body weight), and is involved in the regulation of fat metabolism, as well as in a novel hematopoietic pathway that is required for normal lymphopoiesis. Mutations in this gene have been associated with obesity and pituitary dysfunction. Alternatively spliced transcript variants encoding different isoforms have been described for this gene. It is noteworthy that this gene and LEPROT gene (GeneID:54741) share the same promoter and the first 2 exons, however, encode distinct proteins (PMID:9207021).[provided by RefSeq, Nov 2010]

Genomic Location

Chr 1 Start: 65,420,652 End: 65,641,559 Build: HG19

5' end 3' end

Associated SNPs

1 / 1 detected

SNP	Genotype	Ref. Allele	Variant Allele	Frequency	Status
rs1137101	A G	A	G	58.43%	Detected
Your Genotype AG Reference Allele A Variant Allele G Population Frequency 58.43% Variant Status Variant Detected Database Link NCBI dbSNP

Variant Detected

MC4R GeneCards

The protein encoded by this gene is a membrane-bound receptor and member of the melanocortin receptor family. The encoded protein interacts with adrenocorticotropic and MSH hormones and is mediated by G proteins. This is an intronless gene. Defects in this gene are a cause of autosomal dominant obesity. [provided by RefSeq, Jan 2010]

Genomic Location

Chr 18 Start: 60,371,062 End: 60,372,775 Build: HG19

5' end 3' end

Associated SNPs

1 / 1 detected

SNP	Genotype	Ref. Allele	Variant Allele	Frequency	Status
rs17782313	C T	T	C	24.00%	Detected
Your Genotype CT Reference Allele T Variant Allele C Population Frequency 24.00% Variant Status Variant Detected Database Link NCBI dbSNP

Variant Detected

NPY GeneCards

This gene encodes a neuropeptide that is widely expressed in the central nervous system and influences many physiological processes, including cortical excitability, stress response, food intake, circadian rhythms, and cardiovascular function. The neuropeptide functions through G protein-coupled receptors to inhibit adenylyl cyclase, activate mitogen-activated protein kinase (MAPK), regulate intracellular calcium levels, and activate potassium channels. A polymorphism in this gene resulting in a change of leucine 7 to proline in the signal peptide is associated with elevated cholesterol levels, higher alcohol consumption, and may be a risk factor for various metabolic and cardiovascular diseases. The protein also exhibits antimicrobial activity against bacteria and fungi. [provided by RefSeq, Oct 2014]

Genomic Location

Chr 7 Start: 24,284,188 End: 24,291,862 Build: HG19

5' end 3' end

Associated SNPs

1 / 1 detected

SNP	Genotype	Ref. Allele	Variant Allele	Frequency	Status
rs16147	C T	T	C	51.70%	Detected
Your Genotype CT Reference Allele T Variant Allele C Population Frequency 51.70% Variant Status Variant Detected Database Link NCBI dbSNP

These are the genetic markers (SNPs) analyzed for this trait. Variations detected in your genome are listed under the "Genotype" column. SNPs showing "--" were not identified in your DNA file.

SNP	Chromosome	Genotype	Variant Allele	Frequency
rs1137101	1	AG	G	58.43%
rs16147	7	CT	C	51.70%
rs9939609	16	AT	A	34.01%
rs17782313	18	CT	C	24.00%

DNA Genics reports genotypes based on the 'positive' strand of the human genome reference sequence (build 37/HG19). Other companies may use the opposite strand, so genotypes may need conversion for comparison.

Important Medical Disclaimer

This report is intended for educational and informational purposes only. It is not intended to provide medical advice or be used solely by the customer in the diagnosis, cure, mitigation, treatment, or prevention of any disease or health condition. If you have serious medical conditions, including but not limited to weight-related issues, diabetes, or heart disease, you should not make changes to your diet or exercise routine without consulting your healthcare provider. Under no circumstances should you modify your medication or medical care without professional medical guidance.

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