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GWAS Study

A germline JAK2 SNP is associated with predisposition to the development of JAK2(V617F)-positive myeloproliferative neoplasms.

Kilpivaara O, Mukherjee S, Schram AM et al.

19287384 PubMed ID
GWAS Study Type
3323 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

KO
Kilpivaara O
MS
Mukherjee S
SA
Schram AM
WM
Wadleigh M
MA
Mullally A
EB
Ebert BL
BA
Bass A
MS
Marubayashi S
HA
Heguy A
GG
Garcia-Manero G
KH
Kantarjian H
OK
Offit K
SR
Stone RM
GD
Gilliland DG
KR
Klein RJ
LR
Levine RL
Chapter II

Abstract

Summary of the research findings

Polycythemia vera, essential thrombocythemia and primary myelofibrosis are myeloproliferative neoplasms (MPN) characterized by multilineage clonal hematopoiesis. Given that the identical somatic activating mutation in the JAK2 tyrosine kinase gene (JAK2(V617F)) is observed in most individuals with polycythemia vera, essential thrombocythemia and primary myelofibrosis, there likely are additional genetic events that contribute to the pathogenesis of these phenotypically distinct disorders. Moreover, family members of individuals with MPN are at higher risk for the development of MPN, consistent with the existence of MPN predisposition loci. We hypothesized that germline variation contributes to MPN predisposition and phenotypic pleiotropy. Genome-wide analysis identified an allele in the JAK2 locus (rs10974944) that predisposes to the development of JAK2(V617F)-positive MPN, as well as three previously unknown MPN modifier loci. We found that JAK2(V617F) is preferentially acquired in cis with the predisposition allele. These data suggest that germline variation is an important contributor to MPN phenotype and predisposition.

324 European ancestry cases, 2,999 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

3323
Total Participants
GWAS
Study Type
No
Replicated
European
Ancestry
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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