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GWAS Study

Genome-wide association study identifies human genetic variants associated with fatal outcome from Lassa fever.

Kotliar D, Raju S, Tabrizi S et al.

38326571 PubMed ID
GWAS Study Type
1598 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

KD
Kotliar D
RS
Raju S
TS
Tabrizi S
OI
Odia I
GA
Goba A
MM
Momoh M
SJ
Sandi JD
NP
Nair P
PE
Phelan E
TR
Tariyal R
EP
Eromon PE
MS
Mehta S
RR
Robles-Sikisaka R
SK
Siddle KJ
SM
Stremlau M
JS
Jalloh S
GS
Gire SK
WS
Winnicki S
CB
Chak B
SS
Schaffner SF
PM
Pauthner M
KE
Karlsson EK
CS
Chapin SR
KS
Kennedy SG
BL
Branco LM
KL
Kanneh L
VJ
Vitti JJ
BN
Broodie N
GA
Gladden-Young A
OO
Omoniwa O
JP
Jiang PP
YN
Yozwiak N
HS
Heuklom S
ML
Moses LM
AG
Akpede GO
AD
Asogun DA
RK
Rubins K
KS
Kales S
HA
Happi AN
IC
Iruolagbe CO
DM
Dic-Ijiewere M
IK
Iraoyah K
OO
Osazuwa OO
OA
Okonkwo AK
KS
Kunz S
MJ
McCormick JB
KS
Khan SH
HA
Honko AN
LE
Lander ES
OM
Oldstone MBA
HL
Hensley L
FO
Folarin OA
OS
Okogbenin SA
GS
Günther S
OH
Ollila HM
TR
Tewhey R
OP
Okokhere PO
SJ
Schieffelin JS
AK
Andersen KG
RS
Reilly SK
GD
Grant DS
GR
Garry RF
BK
Barnes KG
HC
Happi CT
SP
Sabeti PC
Chapter II

Abstract

Summary of the research findings

Infection with Lassa virus (LASV) can cause Lassa fever, a haemorrhagic illness with an estimated fatality rate of 29.7%, but causes no or mild symptoms in many individuals. Here, to investigate whether human genetic variation underlies the heterogeneity of LASV infection, we carried out genome-wide association studies (GWAS) as well as seroprevalence surveys, human leukocyte antigen typing and high-throughput variant functional characterization assays. We analysed Lassa fever susceptibility and fatal outcomes in 533 cases of Lassa fever and 1,986 population controls recruited over a 7 year period in Nigeria and Sierra Leone. We detected genome-wide significant variant associations with Lassa fever fatal outcomes near GRM7 and LIF in the Nigerian cohort. We also show that a haplotype bearing signatures of positive selection and overlapping LARGE1, a required LASV entry factor, is associated with decreased risk of Lassa fever in the Nigerian cohort but not in the Sierra Leone cohort. Overall, we identified variants and genes that may impact the risk of severe Lassa fever, demonstrating how GWAS can provide insight into viral pathogenesis.

411 African ancestry cases, 1,187 African ancestry controls

Chapter III

Study Statistics

Key metrics and study information

1598
Total Participants
GWAS
Study Type
No
Replicated
African unspecified
Ancestry
Nigeria, Sierra Leone
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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Analysis In Progress

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