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GWAS Study

A brain-wide genome-wide association study of candidate quantitative trait loci associated with structural and functional phenotypes of pain sensitivity.

Zhang L, Pan Y, Huang G et al.

36864640 PubMed ID
GWAS Study Type
407 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

ZL
Zhang L
PY
Pan Y
HG
Huang G
LZ
Liang Z
LL
Li L
ZM
Zhang M
ZZ
Zhang Z
Chapter II

Abstract

Summary of the research findings

Individual pain sensitivity is modulated by the brain's structural and functional features, but its heritability remains unclear. This paper conducted a brain-wide genome-wide association study (GWAS) to explore the genetic bases of neuroimage phenotypes of pain sensitivity. In total, 432 normal participants were divided into high and low pain sensitivity groups according to the laser quantitative test threshold. Then, the brain's gray matter density (GMD) features correlated with pain sensitivity were identified. Next, GWAS was performed on each GMD phenotype using quality-controlled genotypes. Based on the heatmap and hierarchical clustering results, the right insula was identified for further refined analysis in terms of subregions GMD and resting-state functional connectivity (rs-FC) phenotypes. The results indicate that the right insula GMD in the high sensitivity group is significantly lower than that in the low sensitivity group. Also, the TT/TC group at locus rs187974 has lower right insula GMD than the CC group. Further, loci at gene CYP2D6 may lead to a variation of rs-FC between the right insula and left putamen. In conclusion, our study suggests that the right insula and multiple candidate loci may be importantly involved in pain sensitivity modulation, which may guide the future development of precision pain therapeutics.

407 individuals

Chapter III

Study Statistics

Key metrics and study information

407
Total Participants
GWAS
Study Type
No
Replicated
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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