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GWAS Study

A genome-wide association study identifies IL23R as an inflammatory bowel disease gene.

Duerr RH, Taylor KD, Brant SR et al.

17068223 PubMed ID
GWAS Study Type
2679 Participants
164 Views
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

DR
Duerr RH
TK
Taylor KD
BS
Brant SR
RJ
Rioux JD
SM
Silverberg MS
DM
Daly MJ
SA
Steinhart AH
AC
Abraham C
RM
Regueiro M
GA
Griffiths A
DT
Dassopoulos T
BA
Bitton A
YH
Yang H
TS
Targan S
DL
Datta LW
KE
Kistner EO
SL
Schumm LP
LA
Lee AT
GP
Gregersen PK
BM
Barmada MM
RJ
Rotter JI
ND
Nicolae DL
CJ
Cho JH
Chapter II

Abstract

Summary of the research findings

The inflammatory bowel diseases Crohn's disease and ulcerative colitis are common, chronic disorders that cause abdominal pain, diarrhea, and gastrointestinal bleeding. To identify genetic factors that might contribute to these disorders, we performed a genome-wide association study. We found a highly significant association between Crohn's disease and the IL23R gene on chromosome 1p31, which encodes a subunit of the receptor for the proinflammatory cytokine interleukin-23. An uncommon coding variant (rs11209026, c.1142G>A, p.Arg381Gln) confers strong protection against Crohn's disease, and additional noncoding IL23R variants are independently associated. Replication studies confirmed IL23R associations in independent cohorts of patients with Crohn's disease or ulcerative colitis. These results and previous studies on the proinflammatory role of IL-23 prioritize this signaling pathway as a therapeutic target in inflammatory bowel disease.

547 European ancestry cases, 548 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

2679
Total Participants
GWAS
Study Type
Yes
Replicated
1,151 cases from 595 families, 433 controls
Replication Participants
Other, European
Ancestry
U.S., Canada
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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Analysis In Progress

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