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GWAS Study

Sequencing of 640,000 exomes identifies <i>GPR75</i> variants associated with protection from obesity.

Akbari P, Gilani A, Sosina O et al.

34210852 PubMed ID
GWAS Study Type
645626 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

AP
Akbari P
GA
Gilani A
SO
Sosina O
KJ
Kosmicki JA
KL
Khrimian L
FY
Fang YY
PT
Persaud T
GV
Garcia V
SD
Sun D
LA
Li A
MJ
Mbatchou J
LA
Locke AE
BC
Benner C
VN
Verweij N
LN
Lin N
HS
Hossain S
AK
Agostinucci K
PJ
Pascale JV
DE
Dirice E
DM
Dunn M
KW
Kraus WE
SS
Shah SH
CY
Chen YI
RJ
Rotter JI
RD
Rader DJ
MO
Melander O
SC
Still CD
MT
Mirshahi T
CD
Carey DJ
BJ
Berumen-Campos J
KP
Kuri-Morales P
AJ
Alegre-Díaz J
TJ
Torres JM
EJ
Emberson JR
CR
Collins R
BS
Balasubramanian S
HA
Hawes A
JM
Jones M
ZB
Zambrowicz B
MA
Murphy AJ
PC
Paulding C
CG
Coppola G
OJ
Overton JD
RJ
Reid JG
SA
Shuldiner AR
CM
Cantor M
KH
Kang HM
AG
Abecasis GR
KK
Karalis K
EA
Economides AN
MJ
Marchini J
YG
Yancopoulos GD
SM
Sleeman MW
AJ
Altarejos J
DG
Della Gatta G
TR
Tapia-Conyer R
SM
Schwartzman ML
BA
Baras A
FM
Ferreira MAR
LL
Lotta LA
Chapter II

Abstract

Summary of the research findings

Large-scale human exome sequencing can identify rare protein-coding variants with a large impact on complex traits such as body adiposity. We sequenced the exomes of 645,626 individuals from the United Kingdom, the United States, and Mexico and estimated associations of rare coding variants with body mass index (BMI). We identified 16 genes with an exome-wide significant association with BMI, including those encoding five brain-expressed G protein-coupled receptors (CALCR, MC4R, GIPR, GPR151, and GPR75). Protein-truncating variants in GPR75 were observed in ~4/10,000 sequenced individuals and were associated with 1.8 kilograms per square meter lower BMI and 54% lower odds of obesity in the heterozygous state. Knock out of Gpr75 in mice resulted in resistance to weight gain and improved glycemic control in a high-fat diet model. Inhibition of GPR75 may provide a therapeutic strategy for obesity.

549,780 European ancestry individuals, 95,846 admixed American ancestry individuals

Chapter III

Study Statistics

Key metrics and study information

645626
Total Participants
GWAS
Study Type
No
Replicated
European, Hispanic or Latin American
Ancestry
U.S., U.K., Mexico
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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