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GWAS Study

Genome-wide association study and meta-analysis find that over 40 loci affect risk of type 1 diabetes.

Barrett JC, Clayton DG, Concannon P et al.

19430480 PubMed ID
GWAS Study Type
29838 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

BJ
Barrett JC
CD
Clayton DG
CP
Concannon P
AB
Akolkar B
CJ
Cooper JD
EH
Erlich HA
JC
Julier C
MG
Morahan G
NJ
Nerup J
NC
Nierras C
PV
Plagnol V
PF
Pociot F
SH
Schuilenburg H
SD
Smyth DJ
SH
Stevens H
TJ
Todd JA
WN
Walker NM
RS
Rich SS
Chapter II

Abstract

Summary of the research findings

Type 1 diabetes (T1D) is a common autoimmune disorder that arises from the action of multiple genetic and environmental risk factors. We report the findings of a genome-wide association study of T1D, combined in a meta-analysis with two previously published studies. The total sample set included 7,514 cases and 9,045 reference samples. Forty-one distinct genomic locations provided evidence for association with T1D in the meta-analysis (P < 10(-6)). After excluding previously reported associations, we further tested 27 regions in an independent set of 4,267 cases, 4,463 controls and 2,319 affected sib-pair (ASP) families. Of these, 18 regions were replicated (P < 0.01; overall P < 5 × 10(-8)) and 4 additional regions provided nominal evidence of replication (P < 0.05). The many new candidate genes suggested by these results include IL10, IL19, IL20, GLIS3, CD69 and IL27.

7,514 European ancestry cases, 9,045 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

29838
Total Participants
GWAS
Study Type
Yes
Replicated
4,267 European ancestry cases, 4,670 European ancestry controls, 4,342 European ancestry trios from 2,319 families
Replication Participants
European
Ancestry
U.K., Denmark
Recruitment Country
Chapter IV

AI-Generated Summary

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