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GWAS Study

Genome-wide association meta-analysis of human longevity identifies a novel locus conferring survival beyond 90 years of age.

Deelen J, Beekman M, Uh HW et al.

24688116 PubMed ID
GWAS Study Type
89004 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

DJ
Deelen J
BM
Beekman M
UH
Uh HW
BL
Broer L
AK
Ayers KL
TQ
Tan Q
KY
Kamatani Y
BA
Bennet AM
TR
Tamm R
TS
Trompet S
GD
Guðbjartsson DF
FF
Flachsbart F
RG
Rose G
VA
Viktorin A
FK
Fischer K
NM
Nygaard M
CH
Cordell HJ
CP
Crocco P
VD
van den Akker EB
BS
Böhringer S
HQ
Helmer Q
NC
Nelson CP
SG
Saunders GI
AM
Alver M
AK
Andersen-Ranberg K
BM
Breen ME
VD
van der Breggen R
CA
Caliebe A
CM
Capri M
CE
Cevenini E
CJ
Collerton JC
DS
Dato S
DK
Davies K
FI
Ford I
GJ
Gampe J
GP
Garagnani P
DG
de Geus EJ
HJ
Harrow J
VH
van Heemst D
HB
Heijmans BT
HF
Heinsen FA
HJ
Hottenga JJ
HA
Hofman A
JB
Jeune B
JP
Jonsson PV
LM
Lathrop M
LD
Lechner D
MC
Martin-Ruiz C
MS
Mcnerlan SE
ME
Mihailov E
MA
Montesanto A
MS
Mooijaart SP
MA
Murphy A
NE
Nohr EA
PL
Paternoster L
PI
Postmus I
RF
Rivadeneira F
RO
Ross OA
SS
Salvioli S
SN
Sattar N
SS
Schreiber S
SH
Stefánsson H
SD
Stott DJ
TH
Tiemeier H
UA
Uitterlinden AG
WR
Westendorp RG
WG
Willemsen G
SN
Samani NJ
GP
Galan P
ST
Sørensen TI
BD
Boomsma DI
JJ
Jukema JW
RI
Rea IM
PG
Passarino G
DC
de Craen AJ
CK
Christensen K
NA
Nebel A
SK
Stefánsson K
MA
Metspalu A
MP
Magnusson P
BH
Blanché H
CL
Christiansen L
KT
Kirkwood TB
VD
van Duijn CM
FC
Franceschi C
HJ
Houwing-Duistermaat JJ
SP
Slagboom PE
Chapter II

Abstract

Summary of the research findings

The genetic contribution to the variation in human lifespan is ∼ 25%. Despite the large number of identified disease-susceptibility loci, it is not known which loci influence population mortality. We performed a genome-wide association meta-analysis of 7729 long-lived individuals of European descent (≥ 85 years) and 16 121 younger controls (<65 years) followed by replication in an additional set of 13 060 long-lived individuals and 61 156 controls. In addition, we performed a subset analysis in cases aged ≥ 90 years. We observed genome-wide significant association with longevity, as reflected by survival to ages beyond 90 years, at a novel locus, rs2149954, on chromosome 5q33.3 (OR = 1.10, P = 1.74 × 10(-8)). We also confirmed association of rs4420638 on chromosome 19q13.32 (OR = 0.72, P = 3.40 × 10(-36)), representing the TOMM40/APOE/APOC1 locus. In a prospective meta-analysis (n = 34 103), the minor allele of rs2149954 (T) on chromosome 5q33.3 associates with increased survival (HR = 0.95, P = 0.003). This allele has previously been reported to associate with low blood pressure in middle age. Interestingly, the minor allele (T) associates with decreased cardiovascular mortality risk, independent of blood pressure. We report on the first GWAS-identified longevity locus on chromosome 5q33.3 influencing survival in the general European population. The minor allele of this locus associates with low blood pressure in middle age, although the contribution of this allele to survival may be less dependent on blood pressure. Hence, the pleiotropic mechanisms by which this intragenic variation contributes to lifespan regulation have to be elucidated.

5,406 European ancestry cases, 15,112 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

89004
Total Participants
GWAS
Study Type
Yes
Replicated
7,330 European ancestry cases, 61,156 European ancestry controls
Replication Participants
European
Ancestry
Iceland, Italy, Netherlands, Germany, U.K., Denmark, Sweden, Estonia, France
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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