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GWAS Study

Discovery and refinement of genetic loci associated with cardiometabolic risk using dense imputation maps.

Iotchkova V, Huang J, Morris JA et al.

27668658 PubMed ID
GWAS Study Type
138486 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

IV
Iotchkova V
HJ
Huang J
MJ
Morris JA
JD
Jain D
BC
Barbieri C
WK
Walter K
MJ
Min JL
CL
Chen L
AW
Astle W
CM
Cocca M
DP
Deelen P
EH
Elding H
FA
Farmaki AE
FC
Franklin CS
FM
Franberg M
GT
Gaunt TR
HA
Hofman A
JT
Jiang T
KM
Kleber ME
LG
Lachance G
LJ
Luan J
MG
Malerba G
MA
Matchan A
MD
Mead D
MY
Memari Y
NI
Ntalla I
PK
Panoutsopoulou K
PR
Pazoki R
PJ
Perry JRB
RF
Rivadeneira F
SM
Sabater-Lleal M
SB
Sennblad B
SS
Shin SY
SL
Southam L
TM
Traglia M
VD
van Dijk F
VL
van Leeuwen EM
ZG
Zaza G
ZW
Zhang W
AN
Amin N
BA
Butterworth A
CJ
Chambers JC
DG
Dedoussis G
DA
Dehghan A
FO
Franco OH
FL
Franke L
FM
Frontini M
GG
Gambaro G
GP
Gasparini P
HA
Hamsten A
IA
Issacs A
KJ
Kooner JS
KC
Kooperberg C
LC
Langenberg C
MW
Marz W
SR
Scott RA
SM
Swertz MA
TD
Toniolo D
UA
Uitterlinden AG
VD
van Duijn CM
WH
Watkins H
ZE
Zeggini E
MM
Maurano MT
TN
Timpson NJ
RA
Reiner AP
AP
Auer PL
SN
Soranzo N
Chapter II

Abstract

Summary of the research findings

Large-scale whole-genome sequence data sets offer novel opportunities to identify genetic variation underlying human traits. Here we apply genotype imputation based on whole-genome sequence data from the UK10K and 1000 Genomes Project into 35,981 study participants of European ancestry, followed by association analysis with 20 quantitative cardiometabolic and hematological traits. We describe 17 new associations, including 6 rare (minor allele frequency (MAF) < 1%) or low-frequency (1% < MAF < 5%) variants with platelet count (PLT), red blood cell indices (MCH and MCV) and HDL cholesterol. Applying fine-mapping analysis to 233 known and new loci associated with the 20 traits, we resolve the associations of 59 loci to credible sets of 20 or fewer variants and describe trait enrichments within regions of predicted regulatory function. These findings improve understanding of the allelic architecture of risk factors for cardiometabolic and hematological diseases and provide additional functional insights with the identification of potentially novel biological targets.

up to 35,981 European ancestry individuals

Chapter III

Study Statistics

Key metrics and study information

138486
Total Participants
GWAS
Study Type
Yes
Replicated
up to 102,505 European ancestry individuals
Replication Participants
European
Ancestry
U.S., Germany, U.K., Greece, Italy, Netherlands, Sweden
Recruitment Country
Chapter IV

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