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GWAS Study

Genome-wide association study across European and African American ancestries identifies a SNP in DNMT3B contributing to nicotine dependence.

Hancock DB, Guo Y, Reginsson GW et al.

28972577 PubMed ID
GWAS Study Type
38602 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

HD
Hancock DB
GY
Guo Y
RG
Reginsson GW
GN
Gaddis NC
LS
Lutz SM
SR
Sherva R
LA
Loukola A
MC
Minica CC
MC
Markunas CA
HY
Han Y
YK
Young KA
GD
Gudbjartsson DF
GF
Gu F
MD
McNeil DW
QB
Qaiser B
GC
Glasheen C
OS
Olson S
LM
Landi MT
MP
Madden PAF
FL
Farrer LA
VJ
Vink J
SN
Saccone NL
NM
Neale MC
KH
Kranzler HR
MJ
McKay J
HR
Hung RJ
AC
Amos CI
MM
Marazita ML
BD
Boomsma DI
BT
Baker TB
GJ
Gelernter J
KJ
Kaprio J
CN
Caporaso NE
TT
Thorgeirsson TE
HJ
Hokanson JE
BL
Bierut LJ
SK
Stefansson K
JE
Johnson EO
Chapter II

Abstract

Summary of the research findings

Cigarette smoking is a leading cause of preventable mortality worldwide. Nicotine dependence, which reduces the likelihood of quitting smoking, is a heritable trait with firmly established associations with sequence variants in nicotine acetylcholine receptor genes and at other loci. To search for additional loci, we conducted a genome-wide association study (GWAS) meta-analysis of nicotine dependence, totaling 38,602 smokers (28,677 Europeans/European Americans and 9925 African Americans) across 15 studies. In this largest-ever GWAS meta-analysis for nicotine dependence and the largest-ever cross-ancestry GWAS meta-analysis for any smoking phenotype, we reconfirmed the well-known CHRNA5-CHRNA3-CHRNB4 genes and further yielded a novel association in the DNA methyltransferase gene DNMT3B. The intronic DNMT3B rs910083-C allele (frequency=44-77%) was associated with increased risk of nicotine dependence at P=3.7 × 10-8 (odds ratio (OR)=1.06 and 95% confidence interval (CI)=1.04-1.07 for severe vs mild dependence). The association was independently confirmed in the UK Biobank (N=48,931) using heavy vs never smoking as a proxy phenotype (P=3.6 × 10-4, OR=1.05, and 95% CI=1.02-1.08). Rs910083-C is also associated with increased risk of squamous cell lung carcinoma in the International Lung Cancer Consortium (N=60,586, meta-analysis P=0.0095, OR=1.05, and 95% CI=1.01-1.09). Moreover, rs910083-C was implicated as a cis-methylation quantitative trait locus (QTL) variant associated with higher DNMT3B methylation in fetal brain (N=166, P=2.3 × 10-26) and a cis-expression QTL variant associated with higher DNMT3B expression in adult cerebellum from the Genotype-Tissue Expression project (N=103, P=3.0 × 10-6) and the independent Brain eQTL Almanac (N=134, P=0.028). This novel DNMT3B cis-acting QTL variant highlights the importance of genetically influenced regulation in brain on the risks of nicotine dependence, heavy smoking and consequent lung cancer.

28,677 European ancestry individuals, 9,925 African American individuals

Chapter III

Study Statistics

Key metrics and study information

38602
Total Participants
GWAS
Study Type
No
Replicated
European, African American or Afro-Caribbean
Ancestry
U.S., Netherlands, Finland, Iceland, Italy
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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