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GWAS Study

Genome-wide Analyses Identify KIF5A as a Novel ALS Gene.

Nicolas A, Kenna KP, Renton AE et al.

29566793 PubMed ID
GWAS Study Type
103419 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

NA
Nicolas A
KK
Kenna KP
RA
Renton AE
TN
Ticozzi N
FF
Faghri F
CR
Chia R
DJ
Dominov JA
KB
Kenna BJ
NM
Nalls MA
KP
Keagle P
RA
Rivera AM
VR
van Rheenen W
MN
Murphy NA
VV
van Vugt JJFA
GJ
Geiger JT
VD
Van der Spek RA
PH
Pliner HA
SN
Shankaracharya None
SB
Smith BN
MG
Marangi G
TS
Topp SD
AY
Abramzon Y
GA
Gkazi AS
EJ
Eicher JD
KA
Kenna A
MG
Mora G
CA
Calvo A
ML
Mazzini L
RN
Riva N
MJ
Mandrioli J
CC
Caponnetto C
BS
Battistini S
VP
Volanti P
LB
La Bella V
CF
Conforti FL
BG
Borghero G
MS
Messina S
SI
Simone IL
TF
Trojsi F
SF
Salvi F
LF
Logullo FO
DS
D'Alfonso S
CL
Corrado L
CM
Capasso M
FL
Ferrucci L
MC
Moreno CAM
KS
Kamalakaran S
GD
Goldstein DB
GA
Gitler AD
HT
Harris T
MR
Myers RM
PH
Phatnani H
MR
Musunuri RL
EU
Evani US
AA
Abhyankar A
ZM
Zody MC
KJ
Kaye J
FS
Finkbeiner S
WS
Wyman SK
LA
LeNail A
LL
Lima L
FE
Fraenkel E
SC
Svendsen CN
TL
Thompson LM
VE
Van Eyk JE
BJ
Berry JD
MT
Miller TM
KS
Kolb SJ
CM
Cudkowicz M
BE
Baxi E
BM
Benatar M
TJ
Taylor JP
RE
Rampersaud E
WG
Wu G
WJ
Wuu J
LG
Lauria G
VF
Verde F
FI
Fogh I
TC
Tiloca C
CG
Comi GP
SG
Sorarù G
CC
Cereda C
CP
Corcia P
LH
Laaksovirta H
ML
Myllykangas L
JL
Jansson L
VM
Valori M
EJ
Ealing J
HH
Hamdalla H
RS
Rollinson S
PS
Pickering-Brown S
OR
Orrell RW
SK
Sidle KC
MA
Malaspina A
HJ
Hardy J
SA
Singleton AB
JJ
Johnson JO
AS
Arepalli S
SP
Sapp PC
MD
McKenna-Yasek D
PM
Polak M
AS
Asress S
AS
Al-Sarraj S
KA
King A
TC
Troakes C
VC
Vance C
DB
de Belleroche J
BF
Baas F
TA
Ten Asbroek ALMA
MJ
Muñoz-Blanco JL
HD
Hernandez DG
DJ
Ding J
GJ
Gibbs JR
SS
Scholz SW
FM
Floeter MK
CR
Campbell RH
LF
Landi F
BR
Bowser R
PS
Pulst SM
RJ
Ravits JM
MD
MacGowan DJL
KJ
Kirby J
PE
Pioro EP
PR
Pamphlett R
BJ
Broach J
GG
Gerhard G
DT
Dunckley TL
BC
Brady CB
KN
Kowall NW
TJ
Troncoso JC
LB
Le Ber I
MK
Mouzat K
LS
Lumbroso S
HT
Heiman-Patterson TD
KF
Kamel F
VD
Van Den Bosch L
BR
Baloh RH
ST
Strom TM
MT
Meitinger T
SA
Shatunov A
VE
Van Eijk KR
DC
de Carvalho M
KM
Kooyman M
MB
Middelkoop B
MM
Moisse M
MR
McLaughlin RL
VE
Van Es MA
WM
Weber M
BK
Boylan KB
VB
Van Blitterswijk M
RR
Rademakers R
MK
Morrison KE
BA
Basak AN
MJ
Mora JS
DV
Drory VE
SP
Shaw PJ
TM
Turner MR
TK
Talbot K
HO
Hardiman O
WK
Williams KL
FJ
Fifita JA
NG
Nicholson GA
BI
Blair IP
RG
Rouleau GA
EJ
Esteban-Pérez J
GA
García-Redondo A
AA
Al-Chalabi A
RE
Rogaeva E
ZL
Zinman L
OL
Ostrow LW
MN
Maragakis NJ
RJ
Rothstein JD
SZ
Simmons Z
CJ
Cooper-Knock J
BA
Brice A
GS
Goutman SA
FE
Feldman EL
GS
Gibson SB
TF
Taroni F
RA
Ratti A
GC
Gellera C
VD
Van Damme P
RW
Robberecht W
FP
Fratta P
SM
Sabatelli M
LC
Lunetta C
LA
Ludolph AC
AP
Andersen PM
WJ
Weishaupt JH
CW
Camu W
TJ
Trojanowski JQ
VD
Van Deerlin VM
BR
Brown RH
VD
van den Berg LH
VJ
Veldink JH
HM
Harms MB
GJ
Glass JD
SD
Stone DJ
TP
Tienari P
SV
Silani V
CA
Chiò A
SC
Shaw CE
TB
Traynor BJ
LJ
Landers JE
Chapter II

Abstract

Summary of the research findings

To identify novel genes associated with ALS, we undertook two lines of investigation. We carried out a genome-wide association study comparing 20,806 ALS cases and 59,804 controls. Independently, we performed a rare variant burden analysis comparing 1,138 index familial ALS cases and 19,494 controls. Through both approaches, we identified kinesin family member 5A (KIF5A) as a novel gene associated with ALS. Interestingly, mutations predominantly in the N-terminal motor domain of KIF5A are causative for two neurodegenerative diseases: hereditary spastic paraplegia (SPG10) and Charcot-Marie-Tooth type 2 (CMT2). In contrast, ALS-associated mutations are primarily located at the C-terminal cargo-binding tail domain and patients harboring loss-of-function mutations displayed an extended survival relative to typical ALS cases. Taken together, these results broaden the phenotype spectrum resulting from mutations in KIF5A and strengthen the role of cytoskeletal defects in the pathogenesis of ALS.

20,806 European ancestry cases, 59,804 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

103419
Total Participants
GWAS
Study Type
Yes
Replicated
4,159 cases, 18,650 controls
Replication Participants
European
Ancestry
Belgium, U.S., France, U.K., Italy
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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Analysis In Progress

Our analysis of this publication is currently being prepared. Please check back soon for comprehensive insights into the health and genetic findings discussed in this research.