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GWAS Study

A locus at 7p14.3 predisposes to refractory celiac disease progression from celiac disease.

Hrdlickova B, Mulder CJ, Malamut G et al.

29787419 PubMed ID
GWAS Study Type
2225 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

HB
Hrdlickova B
MC
Mulder CJ
MG
Malamut G
MB
Meresse B
PM
Platteel M
KY
Kamatani Y
RI
Ricaño-Ponce I
VW
van Wanrooij RLJ
ZM
Zorro MM
JB
Jan Bonder M
GJ
Gutierrez-Achury J
CC
Cellier C
ZA
Zhernakova A
NP
Nijeboer P
GP
Galan P
WS
Withoff S
LM
Lathrop M
BG
Bouma G
XR
Xavier RJ
JB
Jabri B
BN
Bensussan NC
WC
Wijmenga C
KV
Kumar V
Chapter II

Abstract

Summary of the research findings

Approximately 5% of patients with celiac disease (CeD) do not respond to a gluten-free diet and progress to refractory celiac disease (RCD), a severe progression that is characterized by infiltration of intraepithelial T lymphocytes. Patients with RCD type II (RCDII) show clonal expansions of intraepithelial T lymphocytes that result in a poor prognosis and a high mortality rate through development of aggressive enteropathy-associated T-cell lymphoma. It is not known whether genetic variations play a role in severe progression of CeD to RCDII.

38 Dutch ancestry cases, 846 Dutch ancestry controls

Chapter III

Study Statistics

Key metrics and study information

2225
Total Participants
GWAS
Study Type
Yes
Replicated
56 European ancestry cases, 1,285 European ancestry controls
Replication Participants
European
Ancestry
Netherlands, France
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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Analysis In Progress

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