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GWAS Study

Genetic variants associated with longitudinal changes in brain structure across the lifespan.

Brouwer RM, Klein M, Grasby KL et al.

35383335 PubMed ID
GWAS Study Type
15100 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

BR
Brouwer RM
KM
Klein M
GK
Grasby KL
SH
Schnack HG
JN
Jahanshad N
TJ
Teeuw J
TS
Thomopoulos SI
SE
Sprooten E
FC
Franz CE
GN
Gogtay N
KW
Kremen WS
PM
Panizzon MS
OL
Olde Loohuis LM
WC
Whelan CD
AM
Aghajani M
AC
Alloza C
AD
Alnæs D
AE
Artiges E
AR
Ayesa-Arriola R
BG
Barker GJ
BM
Bastin ME
BE
Blok E
BE
Bøen E
BI
Breukelaar IA
BJ
Bright JK
BE
Buimer EEL
BR
Bülow R
CD
Cannon DM
CS
Ciufolini S
CN
Crossley NA
DC
Damatac CG
DP
Dazzan P
DM
de Mol CL
DZ
de Zwarte SMC
DS
Desrivières S
DC
Díaz-Caneja CM
DN
Doan NT
DK
Dohm K
FJ
Fröhner JH
GJ
Goltermann J
GA
Grigis A
GD
Grotegerd D
HL
Han LKM
HM
Harris MA
HC
Hartman CA
HS
Heany SJ
HW
Heindel W
HD
Heslenfeld DJ
HS
Hohmann S
IB
Ittermann B
JP
Jansen PR
JJ
Janssen J
JT
Jia T
JJ
Jiang J
JC
Jockwitz C
KT
Karali T
KD
Keeser D
KM
Koevoets MGJC
LR
Lenroot RK
MB
Malchow B
MR
Mandl RCW
MV
Medel V
MS
Meinert S
MC
Morgan CA
MT
Mühleisen TW
NL
Nabulsi L
ON
Opel N
DL
de la Foz VO
OB
Overs BJ
PM
Paillère Martinot ML
RR
Redlich R
MT
Marques TR
RJ
Repple J
RG
Roberts G
RG
Roshchupkin GV
SN
Setiaman N
SE
Shumskaya E
SF
Stein F
SG
Sudre G
TS
Takahashi S
TA
Thalamuthu A
TD
Tordesillas-Gutiérrez D
VD
van der Lugt A
VH
van Haren NEM
WJ
Wardlaw JM
WW
Wen W
WH
Westeneng HJ
WK
Wittfeld K
ZA
Zhu AH
ZA
Zugman A
AN
Armstrong NJ
BG
Bonfiglio G
BJ
Bralten J
DS
Dalvie S
DG
Davies G
DF
Di Forti M
DL
Ding L
DG
Donohoe G
FA
Forstner AJ
GJ
Gonzalez-Peñas J
GJ
Guimaraes JPOFT
HG
Homuth G
HJ
Hottenga JJ
KM
Knol MJ
KJ
Kwok JBJ
LH
Le Hellard S
MK
Mather KA
MY
Milaneschi Y
MD
Morris DW
NM
Nöthen MM
PS
Papiol S
RM
Rietschel M
SM
Santoro ML
SV
Steen VM
SJ
Stein JL
SF
Streit F
TR
Tankard RM
TA
Teumer A
V'
van 't Ent D
VD
van der Meer D
VE
van Eijk KR
VE
Vassos E
VJ
Vázquez-Bourgon J
WS
Witt SH
AH
Adams HHH
AI
Agartz I
AD
Ames D
AK
Amunts K
AO
Andreassen OA
AC
Arango C
BT
Banaschewski T
BB
Baune BT
BS
Belangero SI
BA
Bokde ALW
BD
Boomsma DI
BR
Bressan RA
BH
Brodaty H
BJ
Buitelaar JK
CW
Cahn W
CS
Caspers S
CS
Cichon S
CB
Crespo-Facorro B
CS
Cox SR
DU
Dannlowski U
ET
Elvsåshagen T
ET
Espeseth T
FP
Falkai PG
FS
Fisher SE
FH
Flor H
FJ
Fullerton JM
GH
Garavan H
GP
Gowland PA
GH
Grabe HJ
HT
Hahn T
HA
Heinz A
HM
Hillegers M
HJ
Hoare J
HP
Hoekstra PJ
IM
Ikram MA
JA
Jackowski AP
JA
Jansen A
JE
Jönsson EG
KR
Kahn RS
KT
Kircher T
KM
Korgaonkar MS
KA
Krug A
LH
Lemaitre H
MU
Malt UF
MJ
Martinot JL
MC
McDonald C
MP
Mitchell PB
MR
Muetzel RL
MR
Murray RM
NF
Nees F
NI
Nenadić I
OJ
Oosterlaan J
OR
Ophoff RA
PP
Pan PM
PB
Penninx BWJH
PL
Poustka L
SP
Sachdev PS
SG
Salum GA
SP
Schofield PR
SG
Schumann G
SP
Shaw P
SK
Sim K
SM
Smolka MN
SD
Stein DJ
TJ
Trollor JN
VD
van den Berg LH
VJ
Veldink JH
WH
Walter H
WL
Westlye LT
WR
Whelan R
WT
White T
WM
Wright MJ
MS
Medland SE
FB
Franke B
TP
Thompson PM
HP
Hulshoff Pol HE
Chapter II

Abstract

Summary of the research findings

Human brain structure changes throughout the lifespan. Altered brain growth or rates of decline are implicated in a vast range of psychiatric, developmental and neurodegenerative diseases. In this study, we identified common genetic variants that affect rates of brain growth or atrophy in what is, to our knowledge, the first genome-wide association meta-analysis of changes in brain morphology across the lifespan. Longitudinal magnetic resonance imaging data from 15,640 individuals were used to compute rates of change for 15 brain structures. The most robustly identified genes GPR139, DACH1 and APOE are associated with metabolic processes. We demonstrate global genetic overlap with depression, schizophrenia, cognitive functioning, insomnia, height, body mass index and smoking. Gene set findings implicate both early brain development and neurodegenerative processes in the rates of brain changes. Identifying variants involved in structural brain changes may help to determine biological pathways underlying optimal and dysfunctional brain development and aging.

15,100 European ancestry individuals

Chapter III

Study Statistics

Key metrics and study information

15100
Total Participants
GWAS
Study Type
No
Replicated
European, Other admixed ancestry, Other
Ancestry
U.K.
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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Analysis In Progress

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