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Whole-Exome sequencing analysis identified TMSB10/TRABD2A locus to be associated with carfilzomib-related cardiotoxicity among patients with multiple myeloma.

Tantawy M, Yang G, Algubelli RR et al.

37408649 PubMed ID
GWAS Study Type
228 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

TM
Tantawy M
YG
Yang G
AR
Algubelli RR
DG
DeAvila G
RS
Rubinstein SM
CR
Cornell RF
FM
Fradley MG
SE
Siegel EM
HO
Hampton OA
SA
Silva AS
LD
Lenihan D
SK
Shain KH
BR
Baz RC
GY
Gong Y
Chapter II

Abstract

Summary of the research findings

Proteasome inhibitor Carfilzomib (CFZ) is effective in treating patients with refractory or relapsed multiple myeloma (MM) but has been associated with cardiovascular adverse events (CVAE) such as hypertension, cardiomyopathy, and heart failure. This study aimed to investigate the contribution of germline genetic variants in protein-coding genes in CFZ-CVAE among MM patients using whole-exome sequencing (WES) analysis.

35 European ancestry cases, 193 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

228
Total Participants
GWAS
Study Type
No
Replicated
European, African American or Afro-Caribbean
Ancestry
U.S.
Recruitment Country
Chapter IV

AI-Generated Summary

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