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GWAS Study

Genome-wide association studies highlight novel risk loci for septal defects and left-sided congenital heart defects.

Broberg M, Ampuja M, Jones S et al.

38454350 PubMed ID
GWAS Study Type
882963 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

BM
Broberg M
AM
Ampuja M
JS
Jones S
OT
Ojala T
RO
Rahkonen O
KR
Kivelä R
PJ
Priest J
PA
Palotie A
OH
Ollila HM
HE
Helle E
Chapter II

Abstract

Summary of the research findings

Congenital heart defects (CHD) are structural defects of the heart affecting approximately 1% of newborns. They exhibit low penetrance and non-Mendelian patterns of inheritance as varied and complex traits. While genetic factors are known to play an important role in the development of CHD, the specific genetics remain unknown for the majority of patients. To elucidate the underlying genetic risk, we performed a genome wide association study (GWAS) of CHDs in general and specific CHD subgroups using the FinnGen Release 10 (R10) (N > 393,000), followed by functional fine-mapping through eQTL and co-localization analyses using the GTEx database.

3,506 Finnish ancestry cases, 392,436 Finnish ancestry controls

Chapter III

Study Statistics

Key metrics and study information

882963
Total Participants
GWAS
Study Type
Yes
Replicated
2,156 European ancestry cases, 484,865 European ancestry controls
Replication Participants
European
Ancestry
Finland, U.K.
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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