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GWAS Study

Genome-wide association study and meta-analysis in multiple populations identifies new loci for peanut allergy and establishes C11orf30/EMSY as a genetic risk factor for food allergy.

Asai Y, Eslami A, van Ginkel CD et al.

29030101 PubMed ID
GWAS Study Type
7028 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

AY
Asai Y
EA
Eslami A
VG
van Ginkel CD
AL
Akhabir L
WM
Wan M
EG
Ellis G
BM
Ben-Shoshan M
MD
Martino D
FM
Ferreira MA
AK
Allen K
MB
Mazer B
DG
de Groot H
DJ
de Jong NW
GV
Gerth van Wijk RN
DA
Dubois AEJ
CR
Chin R
CS
Cheuk S
HJ
Hoffman J
JE
Jorgensen E
WJ
Witte JS
MR
Melles RB
HX
Hong X
WX
Wang X
HJ
Hui J
MA
Musk AWB
HM
Hunter M
JA
James AL
KG
Koppelman GH
SA
Sandford AJ
CA
Clarke AE
DD
Daley D
Chapter II

Abstract

Summary of the research findings

Peanut allergy (PA) is a complex disease with both environmental and genetic risk factors. Previously, PA loci were identified in filaggrin (FLG) and HLA in candidate gene studies, and loci in HLA were identified in a genome-wide association study and meta-analysis.

1,068 European ancestry cases, 3,327 European ancestry controls, 514 cases, 2,119 controls

Chapter III

Study Statistics

Key metrics and study information

7028
Total Participants
GWAS
Study Type
No
Replicated
European
Ancestry
U.S., Canada, Australia, Germany
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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Analysis In Progress

Our analysis of this publication is currently being prepared. Please check back soon for comprehensive insights into the health and genetic findings discussed in this research.