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GWAS Study

Variants associating with uterine leiomyoma highlight genetic background shared by various cancers and hormone-related traits.

Rafnar T, Gunnarsson B, Stefansson OA et al.

30194396 PubMed ID
GWAS Study Type
539925 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

RT
Rafnar T
GB
Gunnarsson B
SO
Stefansson OA
SP
Sulem P
IA
Ingason A
FM
Frigge ML
SL
Stefansdottir L
SJ
Sigurdsson JK
TV
Tragante V
SV
Steinthorsdottir V
SU
Styrkarsdottir U
SS
Stacey SN
GJ
Gudmundsson J
AG
Arnadottir GA
OA
Oddsson A
ZF
Zink F
HG
Halldorsson G
SG
Sveinbjornsson G
KR
Kristjansson RP
DO
Davidsson OB
SA
Salvarsdottir A
TA
Thoroddsen A
HE
Helgadottir EA
KK
Kristjansdottir K
IO
Ingthorsson O
GV
Gudmundsson V
GR
Geirsson RT
AR
Arnadottir R
GD
Gudbjartsson DF
MG
Masson G
AF
Asselbergs FW
JJ
Jonasson JG
OK
Olafsson K
TU
Thorsteinsdottir U
HB
Halldorsson BV
TG
Thorleifsson G
SK
Stefansson K
Chapter II

Abstract

Summary of the research findings

Uterine leiomyomas are common benign tumors of the myometrium. We performed a meta-analysis of two genome-wide association studies of leiomyoma in European women (16,595 cases and 523,330 controls), uncovering 21 variants at 16 loci that associate with the disease. Five variants were previously reported to confer risk of various malignant or benign tumors (rs78378222 in TP53, rs10069690 in TERT, rs1800057 and rs1801516 in ATM, and rs7907606 at OBFC1) and four signals are located at established risk loci for hormone-related traits (endometriosis and breast cancer) at 1q36.12 (CDC42/WNT4), 2p25.1 (GREB1), 20p12.3 (MCM8), and 6q26.2 (SYNE1/ESR1). Polygenic score for leiomyoma, computed using UKB data, is significantly correlated with risk of cancer in the Icelandic population. Functional annotation suggests that the non-coding risk variants affect multiple genes, including ESR1. Our results provide insights into the genetic background of leiomyoma that are shared by other benign and malignant tumors and highlight the role of hormones in leiomyoma growth.

16,595 European ancestry cases, 523,330 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

539925
Total Participants
GWAS
Study Type
No
Replicated
European
Ancestry
Iceland, U.K.
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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