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GWAS Study

Two novel pleiotropic loci associated with osteoporosis and abdominal obesity.

Liu L, Yang XL, Zhang H et al.

32239398 PubMed ID
GWAS Study Type
13035 Participants
40 Views
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

LL
Liu L
YX
Yang XL
ZH
Zhang H
ZZ
Zhang ZJ
WX
Wei XT
FG
Feng GJ
LJ
Liu J
PH
Peng HP
HR
Hai R
SH
Shen H
TQ
Tian Q
DH
Deng HW
PY
Pei YF
ZL
Zhang L
Chapter II

Abstract

Summary of the research findings

Aiming to uncover a shared genetic basis of abdominal obesity and osteoporosis, we performed a bivariate GWAS meta-analysis of femoral neck BMD (FNK-BMD) and trunk fat mass adjusted by trunk lean mass (TFMadj) in 11,496 subjects from 6 samples, followed by in silico replication in the large-scale UK Biobank (UKB) cohort. A series of functional investigations were conducted on the identified variants. Bivariate GWAS meta-analysis identified two novel pleiotropic loci 12q15 (lead SNP rs73134637, p = 3.45 × 10-7) and 10p14 (lead SNP rs2892347, p = 2.63 × 10-7) that were suggestively associated and that were replicated in the analyses of related traits in the UKB sample (osteoporosis p = 0.06 and 0.02, BMI p = 0.03 and 4.61 × 10-3, N up to 499,520). Cis-eQTL analysis demonstrated that allele C at rs73134637 was positively associated with IFNG expression in whole blood (N = 369, p = 0.04), and allele A at rs11254759 (10p14, p = 9.49 × 10-7) was negatively associated with PRKCQ expression in visceral adipose tissue (N = 313, p = 0.04) and in lymphocytes (N = 117, p = 0.03). As a proof-of-principle experiment, the function of rs11254759, which is 235 kb 5'-upstream from PRKCQ gene, was investigated by the dual-luciferase reporter assay, which clearly showed that the haplotype carrying rs11254759 regulated PRKCQ expression by upregulating PRKCQ promoter activity (p = 4.60 × 10-7) in an allelic specific manner. Mouse model analysis showed that heterozygous PRKCQ deficient mice presented decreased fat mass compared to wild-type control mice (p = 3.30 × 10-3). Mendelian randomization analysis demonstrated that both FNK-BMD and TFMadj were causally associated with fracture risk (p = 1.26 × 10-23 and 1.18 × 10-11). Our findings may provide useful insights into the genetic association between osteoporosis and abdominal obesity.

10,208 European ancestry individuals, 445 Hispanic individuals, 843 African ancestry individuals, 1,539 East Asian individuals

Chapter III

Study Statistics

Key metrics and study information

13035
Total Participants
GWAS
Study Type
No
Replicated
East Asian, European, African unspecified, Native American, Hispanic or Latin American
Ancestry
China
Recruitment Country
Chapter IV

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