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GWAS Study

Integrative common and rare variant analyses provide insights into the genetic architecture of liver cirrhosis.

Ghouse J, Sveinbjörnsson G, Vujkovic M et al.

38632349 PubMed ID
GWAS Study Type
2039708 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

GJ
Ghouse J
SG
Sveinbjörnsson G
VM
Vujkovic M
SA
Seidelin AS
GH
Gellert-Kristensen H
AG
Ahlberg G
TV
Tragante V
RS
Rand SA
BJ
Brancale J
VS
Vilarinho S
LP
Lundegaard PR
SE
Sørensen E
EC
Erikstrup C
BM
Bruun MT
JB
Jensen BA
BS
Brunak S
BK
Banasik K
UH
Ullum H
VN
Verweij N
LL
Lotta L
BA
Baras A
MT
Mirshahi T
CD
Carey DJ
KD
Kaplan DE
LJ
Lynch J
MT
Morgan T
ST
Schwantes-An TH
DD
Dochtermann DR
PS
Pyarajan S
TP
Tsao PS
LT
Laisk T
MR
Mägi R
KJ
Kozlitina J
TA
Tybjærg-Hansen A
JD
Jones D
KK
Knowlton KU
NL
Nadauld L
FE
Ferkingstad E
BE
Björnsson ES
UM
Ulfarsson MO
Sturluson Á
SP
Sulem P
PO
Pedersen OB
OS
Ostrowski SR
GD
Gudbjartsson DF
SK
Stefansson K
OM
Olesen MS
CK
Chang KM
HH
Holm H
BH
Bundgaard H
SS
Stender S
Chapter II

Abstract

Summary of the research findings

We report a multi-ancestry genome-wide association study on liver cirrhosis and its associated endophenotypes, alanine aminotransferase (ALT) and γ-glutamyl transferase. Using data from 12 cohorts, including 18,265 cases with cirrhosis, 1,782,047 controls, up to 1 million individuals with liver function tests and a validation cohort of 21,689 cases and 617,729 controls, we identify and validate 14 risk associations for cirrhosis. Many variants are located near genes involved in hepatic lipid metabolism. One of these, PNPLA3 p.Ile148Met, interacts with alcohol intake, obesity and diabetes on the risk of cirrhosis and hepatocellular carcinoma (HCC). We develop a polygenic risk score that associates with the progression from cirrhosis to HCC. By focusing on prioritized genes from common variant analyses, we find that rare coding variants in GPAM associate with lower ALT, supporting GPAM as a potential target for therapeutic inhibition. In conclusion, this study provides insights into the genetic underpinnings of cirrhosis.

15,225 European ancestry cases, 1,564,786 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

2039708
Total Participants
GWAS
Study Type
Yes
Replicated
14,041 European ancestry cases, 445,656 European ancestry controls
Replication Participants
European, East Asian, African American or Afro-Caribbean, Hispanic or Latin American
Ancestry
U.S., Finland, Denmark, U.K., Iceland, Germany, Estonia, Japan, Norway
Recruitment Country
Chapter IV

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