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GWAS Study

Sex-specific genetic effects on susceptibility to idiopathic pulmonary fibrosis.

Leavy OC, Goemans AF, Hernandez-Beeftink T et al.

41031098 PubMed ID
GWAS Study Type
27449 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

LO
Leavy OC
GA
Goemans AF
HT
Hernandez-Beeftink T
SA
Stockwell AD
AR
Allen RJ
GB
Guillen-Guio B
AA
Adegunsoye A
BH
Booth HL
FW
Fahy WA
FT
Fingerlin TE
VH
Virk HS
HI
Hall IP
HS
Hart SP
HM
Hill MR
HN
Hirani N
KN
Kaminski N
MS
Ma SF
MR
McAnulty RJ
SX
Sheng XR
MA
Millar AB
MM
Molina-Molina M
NV
Navaratnam V
NM
Neighbors M
PH
Parfrey H
SG
Saini G
SI
Sayers I
SM
Strek ME
TM
Tobin MD
WM
Whyte MKB
ZY
Zhang Y
MT
Maher TM
MP
Molyneaux PL
OJ
Oldham JM
YB
Yaspan BL
FC
Flores C
MF
Martinez F
RC
Reynolds CJ
SD
Schwartz DA
NI
Noth I
JR
Jenkins RG
WL
Wain LV
Chapter II

Abstract

Summary of the research findings

Idiopathic pulmonary fibrosis (IPF) is a chronic lung condition that is more prevalent in males than females. The reasons for this are not fully understood; differing environmental exposures due to historically sex-biased occupations and diagnostic bias are possible explanations. To date, over 20 independent genetic association signals have been reported for IPF susceptibility, but these have been discovered when combining males and females. The objectives of the present study were to assess whether there is a need to consider sex-specific effects when evaluating genetic risk in clinical prediction models for IPF and to test for sex-specific associations with IPF susceptibility.

4,561 European ancestry cases, 22,888 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

27449
Total Participants
GWAS
Study Type
No
Replicated
European
Ancestry
U.S., U.K., Spain
Recruitment Country
Chapter IV

AI-Generated Summary

AI-generated by DNAGENICS

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