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GWAS Study

Genetic variants at 2q24 are associated with susceptibility to type 2 diabetes.

Qi L, Cornelis MC, Kraft P et al.

20418489 PubMed ID
GWAS Study Type
90248 Participants
90 Views
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

QL
Qi L
CM
Cornelis MC
KP
Kraft P
SK
Stanya KJ
LK
Linda Kao WH
PJ
Pankow JS
DJ
Dupuis J
FJ
Florez JC
FC
Fox CS
PG
Paré G
SQ
Sun Q
GC
Girman CJ
LC
Laurie CC
MD
Mirel DB
MT
Manolio TA
CD
Chasman DI
BE
Boerwinkle E
RP
Ridker PM
HD
Hunter DJ
MJ
Meigs JB
LC
Lee CH
HF
Hu FB
VD
van Dam RM
Chapter II

Abstract

Summary of the research findings

To identify type 2 diabetes (T2D) susceptibility loci, we conducted genome-wide association (GWA) scans in nested case-control samples from two prospective cohort studies, including 2591 patients and 3052 controls of European ancestry. Validation was performed in 11 independent GWA studies of 10,870 cases and 73,735 controls. We identified significantly associated variants near RBMS1 and ITGB6 genes at 2q24, best-represented by SNP rs7593730 (combined OR=0.90, 95% CI=0.86-0.93; P=3.7x10(-8)). The frequency of the risk-lowering allele T is 0.23. Variants in this region were nominally related to lower fasting glucose and HOMA-IR in the MAGIC consortium (P<0.05). These data suggest that the 2q24 locus may influence the T2D risk by affecting glucose metabolism and insulin resistance.

2,591 European ancestry cases, 3,052 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

90248
Total Participants
GWAS
Study Type
Yes
Replicated
10,870 European ancestry cases, 73,735 European ancestry controls
Replication Participants
European
Ancestry
Finland, Sweden, U.S., Iceland, Italy, Netherlands, Germany, U.K., Croatia, France
Recruitment Country
Chapter IV

AI-Generated Summary

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