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GWAS Study

Genome-wide association study identifies TF as a significant modifier gene of iron metabolism in HFE hemochromatosis.

de Tayrac M, Roth MP, Jouanolle AM et al.

25457201 PubMed ID
GWAS Study Type
1220 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

DT
de Tayrac M
RM
Roth MP
JA
Jouanolle AM
CH
Coppin H
LG
le Gac G
PA
Piperno A
FC
Férec C
PS
Pelucchi S
SV
Scotet V
BE
Bardou-Jacquet E
RM
Ropert M
BR
Bouvet R
GE
Génin E
MJ
Mosser J
DY
Deugnier Y
Chapter II

Abstract

Summary of the research findings

Background & aims: Hereditary hemochromatosis (HH) is the most common form of genetic iron loading disease. It is mainly related to the homozygous C282Y/C282Y mutation in the HFE gene that is, however, a necessary but not a sufficient condition to develop clinical and even biochemical HH. This suggests that modifier genes are likely involved in the expressivity of the disease. Our aim was to identify such modifier genes.

474 European ancestry cases

Chapter III

Study Statistics

Key metrics and study information

1220
Total Participants
GWAS
Study Type
Yes
Replicated
746 European ancestry cases
Replication Participants
European
Ancestry
Italy, France
Recruitment Country
Chapter IV

AI-Generated Summary

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