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GWAS Study

A GWAS in idiopathic/unexplained infertile men detects a genomic region determining Follicle-stimulating hormone levels.

Schubert M, Pérez Lanuza L, Wöste M et al.

35305013 PubMed ID
GWAS Study Type
1865 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

SM
Schubert M
PL
Pérez Lanuza L
WM
Wöste M
DM
Dugas M
CF
Carmona FD
PR
Palomino-Morales RJ
RY
Rassam Y
HS
Heilmann-Heimbach S
TF
Tüttelmann F
KS
Kliesch S
GJ
Gromoll J
Chapter II

Abstract

Summary of the research findings

Context: Approximately 70% of infertile men are diagnosed with idiopathic (abnormal semen parameters) or unexplained (normozoospermia) infertility, with the common feature of lacking etiologic factors. Follicle-stimulating hormone (FSH) is essential for initiation and maintenance of spermatogenesis. Certain single-nucleotide variations (SNVs; formerly single-nucleotide polymorphisms [SNPs]) (ie, FSHB c.-211G > T, FSHR c.2039A > G) are associated with FSH, testicular volume, and spermatogenesis. It is unknown to what extent other variants are associated with FSH levels and therewith resemble causative factors for infertility.

742 European ancestry individuals

Chapter III

Study Statistics

Key metrics and study information

1865
Total Participants
GWAS
Study Type
Yes
Replicated
1,123 European ancestry individuals
Replication Participants
European
Ancestry
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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