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Admixture-mapping analysis reveals genetic determinants of the human plasma proteome.

Cruz Daniel E, DE Deng, Shuliang S et al.

41069105 PubMed ID
33 Authors
2026-01-15 Published
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

CD
Cruz Daniel E
DD
DE Deng
SS
Shuliang S
TU
Tahir Usman A
UC
UA Chen
ZZ
Zsu-Zsu ZZ
BM
Benson Mark D
MT
MD Tuftin
BB
Bjoernar B
CJ
Chen Jiawen
JF
J Farrell
LL
Laurie L
SD
Shen Dongxiao
DM
D Meyer
MM
Mariah M
LE
Lange Ethan
EG
E Gao
YY
Yan Y
HM
Hall Michael E
MT
ME Tracy
RP
Russell P RP
RS
Rich Stephen S
ST
SS Taylor
KK
Kent K
MA
Manichaikul Ani
AR
A Rotter
JI
Jerome I JI
ST
Sofer Tamar
TW
T Wilson
JG
James G JG
GR
Gerszten Robert E
RR
RE Raffield
LM
Laura M LM
Chapter II

Abstract

Summary of the research findings

Protein profiling and genetic findings can be integrated to define the genetic architecture of the circulating proteome in chronic diseases. Most self-identified African American (AA) individuals have both African and European genetic ancestry. Admixture mapping can detect genomic association regions in which causal variants exist with substantial differences in allele frequency or effect sizes between genetic ancestries. We performed admixture mapping of the circulating proteome in 1,989 participants from the Jackson Heart Study (JHS), investigating the relation of local African ancestry within genomic regions with levels of circulating proteins. We conditioned protein-local ancestry association models on variants previously found to be associated with those proteins in genome-wide association studies (GWASs). We replicated findings in 196 AA participants from the Multi-Ethnic Study of Atherosclerosis (MESA). 62 proteins were associated with local African ancestry. 21 of 62 remained statistically significant after conditioning on protein-associated variants observed in previous GWASs. 48 of 54 available protein-local ancestry associations were replicated in the MESA. Proteins associated with local African ancestry included chemokines, factors associated with vascular biology and inflammation, and other biologically interesting proteins. Admixture associations unexplained by previously reported protein-associated variants in conditional analysis suggest the existence of causal variants missed by standard GWAS techniques.

Chapter III

Analysis

Comprehensive review of ancestry and genetic findings

Important Disclaimer: This review has been performed semi-automatically and is provided for informational purposes only. While we strive for accuracy, this analysis may contain errors, omissions, or misinterpretations of the original research. DNA Genics disclaims all liability for any inaccuracies, errors, or consequences arising from the use of this information. Users should independently verify all information and consult original research publications before making any decisions based on this content. This analysis is not intended as a substitute for professional scientific review or medical advice.

Summary

Key Findings

Ancestry Insights

Traits Analysis

Historical Context

Scientific Assessment