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GWAS Study

Enhanced Contribution of HLA in Pediatric Onset Ulcerative Colitis.

Venkateswaran S, Prince J, Cutler DJ et al.

29562276 PubMed ID
GWAS Study Type
2565 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

VS
Venkateswaran S
PJ
Prince J
CD
Cutler DJ
MU
Marigorta UM
OD
Okou DT
PS
Prahalad S
MD
Mack D
BB
Boyle B
WT
Walters T
GA
Griffiths A
SC
Sauer CG
LN
LeLeiko N
KD
Keljo D
MJ
Markowitz J
BS
Baker SS
RJ
Rosh J
PM
Pfefferkorn M
HM
Heyman MB
PA
Patel A
OA
Otley A
BR
Baldassano R
NJ
Noe J
RP
Rufo P
OM
Oliva-Hemker M
DS
Davis S
ZM
Zwick ME
GG
Gibson G
DL
Denson LA
HJ
Hyams J
KS
Kugathasan S
Chapter II

Abstract

Summary of the research findings

The genetic contributions to pediatric onset ulcerative colitis (UC), characterized by severe disease and extensive colonic involvement, are largely unknown. In adult onset UC, Genome Wide Association Study (GWAS) has identified numerous loci, most of which have a modest susceptibility risk (OR 0.84-1.14), with the exception of the human leukocyte antigen (HLA) region on Chromosome 6 (OR 3.59).

466 European ancestry cases, 2,099 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

2565
Total Participants
GWAS
Study Type
No
Replicated
European
Ancestry
U.S.
Recruitment Country
Chapter IV

AI-Generated Summary

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