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Genetic Regulation of PCSK9 (Proprotein Convertase Subtilisin/Kexin Type 9) Plasma Levels and Its Impact on Atherosclerotic Vascular Disease Phenotypes.

Pott J, Schlegel V, Teren A et al.

29748315 PubMed ID
GWAS Study Type
3290 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

PJ
Pott J
SV
Schlegel V
TA
Teren A
HK
Horn K
KH
Kirsten H
BC
Bluecher C
KJ
Kratzsch J
LM
Loeffler M
TJ
Thiery J
BR
Burkhardt R
SM
Scholz M
Chapter II

Abstract

Summary of the research findings

Inhibition of PCSK9 (proprotein convertase subtilisin/kexin type 9) is a novel strategy to treat hypercholesterolemia and reduce cardiovascular events. However, the potential role of circulating plasma PCSK9 concentrations as a diagnostic and predictive biomarker remains uncertain as of now. Here, we aimed to identify genetic variants associated with plasma PCSK9 and investigate possible causal effects on atherosclerotic vascular disease phenotypes.

2,022 European ancestry individuals without statin treatment, 1,268 European ancestry individuals with statin treatment

Chapter III

Study Statistics

Key metrics and study information

3290
Total Participants
GWAS
Study Type
No
Replicated
European
Ancestry
Germany
Recruitment Country
Chapter IV

AI-Generated Summary

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