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Transethnic Genome-Wide Association Study Provides Insights in the Genetic Architecture and Heritability of Long QT Syndrome.

Lahrouchi N, Tadros R, Crotti L et al.

32429735 PubMed ID
GWAS Study Type
11492 Participants
92 Views
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

LN
Lahrouchi N
TR
Tadros R
CL
Crotti L
MY
Mizusawa Y
PP
Postema PG
BL
Beekman L
WR
Walsh R
HK
Hasegawa K
BJ
Barc J
EM
Ernsting M
TK
Turkowski KL
MA
Mazzanti A
BB
Beckmann BM
SK
Shimamoto K
DU
Diamant UB
WY
Wijeyeratne YD
KY
Kucho Y
RT
Robyns T
IT
Ishikawa T
AE
Arbelo E
CM
Christiansen M
WA
Winbo A
JR
Jabbari R
LS
Lubitz SA
SJ
Steinfurt J
RB
Rudic B
LB
Loeys B
SM
Shoemaker MB
WP
Weeke PE
PR
Pfeiffer R
DB
Davies B
AA
Andorin A
HN
Hofman N
DF
Dagradi F
PM
Pedrazzini M
TD
Tester DJ
BJ
Bos JM
SG
Sarquella-Brugada G
Campuzano Ó
PP
Platonov PG
SB
Stallmeyer B
ZS
Zumhagen S
NE
Nannenberg EA
VJ
Veldink JH
VD
van den Berg LH
AA
Al-Chalabi A
SC
Shaw CE
SP
Shaw PJ
MK
Morrison KE
AP
Andersen PM
MM
Müller-Nurasyid M
CD
Cusi D
BC
Barlassina C
GP
Galan P
LM
Lathrop M
MM
Munter M
WT
Werge T
RM
Ribasés M
AT
Aung T
KC
Khor CC
OM
Ozaki M
LP
Lichtner P
MT
Meitinger T
VT
van Tintelen JP
HY
Hoedemaekers Y
DI
Denjoy I
LA
Leenhardt A
NC
Napolitano C
SW
Shimizu W
SJ
Schott JJ
GJ
Gourraud JB
MT
Makiyama T
OS
Ohno S
IH
Itoh H
KA
Krahn AD
AC
Antzelevitch C
RD
Roden DM
SJ
Saenen J
BM
Borggrefe M
OK
Odening KE
EP
Ellinor PT
TJ
Tfelt-Hansen J
SJ
Skinner JR
VD
van den Berg MP
OM
Olesen MS
BJ
Brugada J
BR
Brugada R
MN
Makita N
BJ
Breckpot J
YM
Yoshinaga M
BE
Behr ER
RA
Rydberg A
AT
Aiba T
KS
Kääb S
PS
Priori SG
GP
Guicheney P
TH
Tan HL
NC
Newton-Cheh C
AM
Ackerman MJ
SP
Schwartz PJ
SE
Schulze-Bahr E
PV
Probst V
HM
Horie M
WA
Wilde AA
TM
Tanck MWT
BC
Bezzina CR
Chapter II

Abstract

Summary of the research findings

Long QT syndrome (LQTS) is a rare genetic disorder and a major preventable cause of sudden cardiac death in the young. A causal rare genetic variant with large effect size is identified in up to 80% of probands (genotype positive) and cascade family screening shows incomplete penetrance of genetic variants. Furthermore, a proportion of cases meeting diagnostic criteria for LQTS remain genetically elusive despite genetic testing of established genes (genotype negative). These observations raise the possibility that common genetic variants with small effect size contribute to the clinical picture of LQTS. This study aimed to characterize and quantify the contribution of common genetic variation to LQTS disease susceptibility.

1,238 European ancestry cases, 8,219 European ancestry controls, 418 Japanese cases, 1,617 Japanese controls

Chapter III

Study Statistics

Key metrics and study information

11492
Total Participants
GWAS
Study Type
No
Replicated
European, East Asian
Ancestry
Belgium, Denmark, France, Germany, Italy, Netherlands, Spain, U.K., U.S., New Zealand, Japan
Recruitment Country
Chapter IV

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