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GWAS Study

Genome-wide analysis identifies novel susceptibility loci for myocardial infarction.

Hartiala JA, Han Y, Jia Q et al.

33532862 PubMed ID
GWAS Study Type
639221 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

HJ
Hartiala JA
HY
Han Y
JQ
Jia Q
HJ
Hilser JR
HP
Huang P
GJ
Gukasyan J
SW
Schwartzman WS
CZ
Cai Z
BS
Biswas S
TD
Trégouët DA
SN
Smith NL
SM
Seldin M
PC
Pan C
MM
Mehrabian M
LA
Lusis AJ
BP
Bazeley P
SY
Sun YV
LC
Liu C
QA
Quyyumi AA
SM
Scholz M
TJ
Thiery J
DG
Delgado GE
KM
Kleber ME
MW
März W
HL
Howe LJ
AF
Asselbergs FW
VV
van Vugt M
VG
Vlachojannis GJ
PR
Patel RS
LL
Lyytikäinen LP
KM
Kähönen M
LT
Lehtimäki T
NT
Nieminen TVM
KP
Kuukasjärvi P
LJ
Laurikka JO
CX
Chang X
HC
Heng CK
JR
Jiang R
KW
Kraus WE
HE
Hauser ER
FJ
Ferguson JF
RM
Reilly MP
IK
Ito K
KS
Koyama S
KY
Kamatani Y
KI
Komuro I
SL
Stolze LK
RC
Romanoski CE
KM
Khan MD
TA
Turner AW
MC
Miller CL
AR
Aherrahrou R
CM
Civelek M
ML
Ma L
BJ
Björkegren JLM
KS
Kumar SR
TW
Tang WHW
HS
Hazen SL
AH
Allayee H
Chapter II

Abstract

Summary of the research findings

Aims: While most patients with myocardial infarction (MI) have underlying coronary atherosclerosis, not all patients with coronary artery disease (CAD) develop MI. We sought to address the hypothesis that some of the genetic factors which establish atherosclerosis may be distinct from those that predispose to vulnerable plaques and thrombus formation.

14,825 European ancestry cases, 44,000 European and unknown ancestry cases, 2,680 cases, 380,970 European ancestry controls, 123,504 European and unknown ancestry controls, 73,242 controls

Chapter III

Study Statistics

Key metrics and study information

639221
Total Participants
GWAS
Study Type
No
Replicated
European, NR, European
Ancestry
U.K.
Recruitment Country
Chapter IV

AI-Generated Summary

AI-generated by DNAGENICS

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