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Colocalization analysis of pancreas eQTLs with risk loci from alcoholic and novel non-alcoholic chronic pancreatitis GWAS suggests potential disease causing mechanisms.

Schmidt AW, Kühnapfel A, Kirsten H et al.

35331647 PubMed ID
GWAS Study Type
6624 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

SA
Schmidt AW
KA
Kühnapfel A
KH
Kirsten H
GH
Grallert H
HC
Hellerbrand C
KF
Kiefer F
MK
Mann K
MS
Mueller S
NM
Nöthen MM
PA
Peters A
RM
Ridinger M
FJ
Frank J
RM
Rietschel M
SN
Soranzo N
SM
Soyka M
WN
Wodarz N
MG
Malerba G
GG
Gambaro G
GC
Gieger C
SM
Scholz M
KS
Krug S
MP
Michl P
EM
Ewers M
WH
Witt H
LH
Laumen H
RJ
Rosendahl J
Chapter II

Abstract

Summary of the research findings

Previous genome-wide association studies (GWAS) identified genome-wide significant risk loci in chronic pancreatitis and investigated underlying disease causing mechanisms by simple overlaps with expression quantitative trait loci (eQTLs), a procedure which may often result in false positive conclusions.

584 European ancestry cases, 6,040 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

6624
Total Participants
GWAS
Study Type
No
Replicated
European
Ancestry
Chapter IV

AI-Generated Summary

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