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GWAS Study

Genetic analysis of lung cancer and the germline impact on somatic mutation burden.

Gabriel AAG, Atkins JR, Penha RCC et al.

35511172 PubMed ID
GWAS Study Type
329946 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

GA
Gabriel AAG
AJ
Atkins JR
PR
Penha RCC
SK
Smith-Byrne K
GV
Gaborieau V
VC
Voegele C
AB
Abedi-Ardekani B
MM
Milojevic M
OR
Olaso R
MV
Meyer V
BA
Boland A
DJ
Deleuze JF
ZD
Zaridze D
MA
Mukeriya A
SB
Swiatkowska B
JV
Janout V
SM
Schejbalová M
MD
Mates D
SJ
Stojšić J
OM
Ognjanovic M
WJ
Witte JS
RS
Rashkin SR
KL
Kachuri L
HR
Hung RJ
KS
Kar S
BP
Brennan P
SA
Sertier AS
FA
Ferrari A
VA
Viari A
JM
Johansson M
AC
Amos CI
FM
Foll M
MJ
McKay JD
Chapter II

Abstract

Summary of the research findings

Germline genetic variation contributes to lung cancer (LC) susceptibility. Previous genome-wide association studies (GWAS) have implicated susceptibility loci involved in smoking behaviors and DNA repair genes, but further work is required to identify susceptibility variants.

29,266 European ancestry lung cancer cases, 48,843 European ancestry individuals with family history of lung cancer, 251,837 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

329946
Total Participants
GWAS
Study Type
No
Replicated
European
Ancestry
U.K.
Recruitment Country
Chapter IV

AI-Generated Summary

AI-generated by DNAGENICS

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