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GWAS Study

Genome-wide association study and Mendelian randomization analyses provide insights into the causes of early-onset colorectal cancer.

Laskar RS, Qu C, Huyghe JR et al.

38408508 PubMed ID
GWAS Study Type
72005 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

LR
Laskar RS
QC
Qu C
HJ
Huyghe JR
HT
Harrison T
HR
Hayes RB
CY
Cao Y
CP
Campbell PT
SR
Steinfelder R
TF
Talukdar FR
BH
Brenner H
OS
Ogino S
BS
Brendt S
BD
Bishop DT
BD
Buchanan DD
CA
Chan AT
CM
Cotterchio M
GS
Gruber SB
GA
Gsur A
VG
van Guelpen B
JM
Jenkins MA
KT
Keku TO
LB
Lynch BM
LM
Le Marchand L
MR
Martin RM
MK
McCarthy K
MV
Moreno V
PR
Pearlman R
SM
Song M
TK
Tsilidis KK
VP
Vodička P
WM
Woods MO
WK
Wu K
HL
Hsu L
GM
Gunter MJ
PU
Peters U
MN
Murphy N
Chapter II

Abstract

Summary of the research findings

The incidence of early-onset colorectal cancer (EOCRC; diagnosed <50 years of age) is rising globally; however, the causes underlying this trend are largely unknown. CRC has strong genetic and environmental determinants, yet common genetic variants and causal modifiable risk factors underlying EOCRC are unknown. We conducted the first EOCRC-specific genome-wide association study (GWAS) and Mendelian randomization (MR) analyses to explore germline genetic and causal modifiable risk factors associated with EOCRC.

6,176 European ancestry cases, 65,829 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

72005
Total Participants
GWAS
Study Type
No
Replicated
European
Ancestry
Puerto Rico, Spain, Canada, Netherlands, Austria, Czech Republic, Sweden, U.S., Finland, Italy, U.K., Israel, Australia, France, Germany
Recruitment Country
Chapter IV

AI-Generated Summary

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