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A genome-wide association study identifies the GPM6A locus associated with age at onset in ALS.

Nakamura R, Tohnai G, Atsuta N et al.

41350806 PubMed ID
GWAS Study Type
2015 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

NR
Nakamura R
TG
Tohnai G
AN
Atsuta N
MY
Matsuda Y
MS
Morimoto S
ID
Ito D
KM
Katsuno M
IY
Izumi Y
MM
Morita M
II
Iwata I
YI
Yabe I
NT
Nakazato T
HN
Hattori N
HT
Hirayama T
KO
Kano O
TA
Tamura A
SN
Suzuki N
AM
Aoki M
SK
Shibuya K
KS
Kuwabara S
OM
Oda M
HR
Hashimoto R
AI
Aiba I
IT
Ishihara T
OO
Onodera O
YT
Yamashita T
IH
Ishiura H
BK
Bokuda K
ST
Shimizu T
IY
Ikeda Y
HK
Hasegawa K
TF
Tanaka F
YT
Yokota T
KK
Kanai K
NY
Noto YI
KR
Kaji R
WH
Watanabe H
KT
Konishi T
HM
Hasegawa M
FH
Fukaya H
NJ
Niwa JI
DM
Doyu M
OY
Okada Y
NS
Nakamura S
OF
Ozawa F
OH
Okano H
NM
Nakatochi M
SG
Sobue G
Chapter II

Abstract

Summary of the research findings

Amyotrophic lateral sclerosis (ALS) exhibits considerable clinical variability, such as differences in age at onset (AAO). Multiple factors, including genetic factors, may underlie this variability; however, the specific determinants remain unclear. To identify genes affecting AAO, we have conducted a genome-wide association study in Japanese patients with ALS (discovery cohort: n = 1808; replication cohort: n = 207). Here, we show that the minor A allele of rs113161727 at the ADAM29-GPM6A locus is associated with a younger AAO in the discovery cohort (effect, -4.27 years; p = 4.60 × 10-8); this finding has been confirmed in the replication cohort (p = 0.0068) and meta-analysis (p = 1.08 × 10-9). Among 65 ALS patients with a SOD1 mutation, the AAO has been found to be 10.2 years younger in those with the A allele than in those without it (p = 0.002). This variant correlates with GPM6A upregulation in iPSC-derived motor neurons, suggesting GPM6A as a candidate AAO modifier. Overall, our study highlights the impact of genetic modifiers on ALS heterogeneity and provides a potential target for delaying disease onset.

2,015 Japanese ancestry cases

Chapter III

Study Statistics

Key metrics and study information

2015
Total Participants
GWAS
Study Type
No
Replicated
East Asian
Ancestry
Japan
Recruitment Country
Chapter IV

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