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GWAS Study

Trait-stratified genome-wide association study identifies novel and diverse genetic associations with serologic and cytokine phenotypes in systemic lupus erythematosus.

Kariuki SN, Franek BS, Kumar AA et al.

20659327 PubMed ID
GWAS Study Type
642 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

KS
Kariuki SN
FB
Franek BS
KA
Kumar AA
AJ
Arrington J
MR
Mikolaitis RA
UT
Utset TO
JM
Jolly M
CM
Crow MK
SA
Skol AD
NT
Niewold TB
Chapter II

Abstract

Summary of the research findings

Introduction: Systemic lupus erythematosus (SLE) is a highly heterogeneous disorder, characterized by differences in autoantibody profile, serum cytokines, and clinical manifestations. SLE-associated autoantibodies and high serum interferon alpha (IFN-α) are important heritable phenotypes in SLE which are correlated with each other, and play a role in disease pathogenesis. These two heritable risk factors are shared between ancestral backgrounds. The aim of the study was to detect genetic factors associated with autoantibody profiles and serum IFN-α in SLE.

44 European, African American, Hispanic American, and Asian American high IFN-alpha cases, 60 European, African American, Hispanic American, and Asian American low IFN-alpha cases

Chapter III

Study Statistics

Key metrics and study information

642
Total Participants
GWAS
Study Type
Yes
Replicated
280 African American cases, 173 European ancestry cases, 85 Hispanic American cases
Replication Participants
Hispanic or Latin American, African American or Afro-Caribbean, Asian unspecified, European
Ancestry
U.S.
Recruitment Country
Chapter IV

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