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GWAS Study

High-density genotyping of immune loci in Koreans and Europeans identifies eight new rheumatoid arthritis risk loci.

Kim K, Bang SY, Lee HS et al.

24532676 PubMed ID
GWAS Study Type
55089 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

KK
Kim K
BS
Bang SY
LH
Lee HS
CS
Cho SK
CC
Choi CB
SY
Sung YK
KT
Kim TH
JJ
Jun JB
YD
Yoo DH
KY
Kang YM
KS
Kim SK
SC
Suh CH
SS
Shim SC
LS
Lee SS
LJ
Lee J
CW
Chung WT
CJ
Choe JY
SH
Shin HD
LJ
Lee JY
HB
Han BG
NS
Nath SK
ES
Eyre S
BJ
Bowes J
PD
Pappas DA
KJ
Kremer JM
GM
Gonzalez-Gay MA
RL
Rodriguez-Rodriguez L
ÄL
Ärlestig L
OY
Okada Y
DD
Diogo D
LK
Liao KP
KE
Karlson EW
RS
Raychaudhuri S
RS
Rantapää-Dahlqvist S
MJ
Martin J
KL
Klareskog L
PL
Padyukov L
GP
Gregersen PK
WJ
Worthington J
GJ
Greenberg JD
PR
Plenge RM
BS
Bae SC
Chapter II

Abstract

Summary of the research findings

Objective: A highly polygenic aetiology and high degree of allele-sharing between ancestries have been well elucidated in genetic studies of rheumatoid arthritis. Recently, the high-density genotyping array Immunochip for immune disease loci identified 14 new rheumatoid arthritis risk loci among individuals of European ancestry. Here, we aimed to identify new rheumatoid arthritis risk loci using Korean-specific Immunochip data.

2,234 Korean ancestry cases, 7,065 Korean ancestry controls, 10,288 European ancestry cases, 35,502 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

55089
Total Participants
GWAS
Study Type
No
Replicated
East Asian, European
Ancestry
Canada, U.S., Netherlands, Sweden, U.K., Spain
Recruitment Country
Chapter IV

AI-Generated Summary

AI-generated by DNAGENICS

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