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CYP24A1 variant modifies the association between use of oestrogen plus progestogen therapy and colorectal cancer risk.

Garcia-Albeniz X, Rudolph A, Hutter C et al.

26766742 PubMed ID
GWAS Study Type
3384 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

GX
Garcia-Albeniz X
RA
Rudolph A
HC
Hutter C
WE
White E
LY
Lin Y
RS
Rosse SA
FJ
Figueiredo JC
HT
Harrison TA
JS
Jiao S
BH
Brenner H
CG
Casey G
HT
Hudson TJ
TM
Thornquist M
LM
Le Marchand L
PJ
Potter J
SM
Slattery ML
ZB
Zanke B
BJ
Baron JA
CB
Caan BJ
CS
Chanock SJ
BS
Berndt SI
SD
Stelling D
FC
Fuchs CS
HM
Hoffmeister M
BK
Butterbach K
DM
Du M
JG
James Gauderman W
GM
Gunter MJ
LM
Lemire M
OS
Ogino S
LJ
Lin J
HR
Hayes RB
HR
Haile RW
SR
Schoen RE
WG
Warnick GS
JM
Jenkins MA
TS
Thibodeau SN
SF
Schumacher FR
LN
Lindor NM
KL
Kolonel LN
HJ
Hopper JL
GJ
Gong J
SD
Seminara D
PB
Pflugeisen BM
UC
Ulrich CM
QC
Qu C
DD
Duggan D
CM
Cotterchio M
CP
Campbell PT
CC
Carlson CS
NP
Newcomb PA
GE
Giovannucci E
HL
Hsu L
CA
Chan AT
PU
Peters U
CJ
Chang-Claude J
Chapter II

Abstract

Summary of the research findings

Menopausal hormone therapy (MHT) use has been consistently associated with a decreased risk of colorectal cancer (CRC) in women. Our aim was to use a genome-wide gene-environment interaction analysis to identify genetic modifiers of CRC risk associated with use of MHT.

1,436 European ancestry postmenopausal cases, 1,948 European ancestry postmenopausal controls

Chapter III

Study Statistics

Key metrics and study information

3384
Total Participants
GWAS
Study Type
No
Replicated
European
Ancestry
U.S., Australia, Canada, Germany
Recruitment Country
Chapter IV

AI-Generated Summary

AI-generated by DNAGENICS

Independent AI summary of health and genetic findings from the published study

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