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GWAS Study

Common variants in MMP20 at 11q22.2 predispose to 11q deletion and neuroblastoma risk.

Chang X, Zhao Y, Hou C et al.

28924153 PubMed ID
GWAS Study Type
7168 Participants
75 Views
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

CX
Chang X
ZY
Zhao Y
HC
Hou C
GJ
Glessner J
ML
McDaniel L
DM
Diamond MA
TK
Thomas K
LJ
Li J
WZ
Wei Z
LY
Liu Y
GY
Guo Y
MF
Mentch FD
QH
Qiu H
KC
Kim C
EP
Evans P
VZ
Vaksman Z
DS
Diskin SJ
AE
Attiyeh EF
SP
Sleiman P
MJ
Maris JM
HH
Hakonarson H
Chapter II

Abstract

Summary of the research findings

MYCN amplification and 11q deletion are two inversely correlated prognostic factors of poor outcome in neuroblastoma. Here we identify common variants at 11q22.2 within MMP20 that associate with neuroblastoma cases harboring 11q deletion (rs10895322), using GWAS in 113 European-American cases and 5109 ancestry-matched controls. The association is replicated in 44 independent cases and 1902 controls. Our study yields novel insights into the genetic underpinnings of neuroblastoma, demonstrating that the inherited common variants reported contribute to the origin of intra-tumor genetic heterogeneity in neuroblastoma.Chromosomal abnormalities such as 11q deletion are associated with poor prognosis in neuroblastoma. Here, the authors perform a genome-wide association study and identify an association between a variant within a Matrix metalloproteinase (MMP) gene member, MMP20, and 11q-deletion subtype neuroblastoma.

113 European ancestry cases, 5,109 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

7168
Total Participants
GWAS
Study Type
Yes
Replicated
44 European ancestry cases, 1,902 European ancestry controls
Replication Participants
European
Ancestry
U.S.
Recruitment Country
Chapter IV

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