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Genetic variants associated with susceptibility to idiopathic pulmonary fibrosis in people of European ancestry: a genome-wide association study.

Allen RJ, Porte J, Braybrooke R et al.

29066090 PubMed ID
GWAS Study Type
11321 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

AR
Allen RJ
PJ
Porte J
BR
Braybrooke R
FC
Flores C
FT
Fingerlin TE
OJ
Oldham JM
GB
Guillen-Guio B
MS
Ma SF
OT
Okamoto T
JA
John AE
OM
Obeidat M
YI
Yang IV
HA
Henry A
HR
Hubbard RB
NV
Navaratnam V
SG
Saini G
TN
Thompson N
BH
Booth HL
HS
Hart SP
HM
Hill MR
HN
Hirani N
MT
Maher TM
MR
McAnulty RJ
MA
Millar AB
MP
Molyneaux PL
PH
Parfrey H
RD
Rassl DM
WM
Whyte MKB
FW
Fahy WA
MR
Marshall RP
OE
Oballa E
BY
Bossé Y
ND
Nickle DC
SD
Sin DD
TW
Timens W
SN
Shrine N
SI
Sayers I
HI
Hall IP
NI
Noth I
SD
Schwartz DA
TM
Tobin MD
WL
Wain LV
JR
Jenkins RG
Chapter II

Abstract

Summary of the research findings

Idiopathic pulmonary fibrosis (IPF) is a chronic progressive lung disease with high mortality, uncertain cause, and few treatment options. Studies have identified a significant genetic risk associated with the development of IPF; however, mechanisms by which genetic risk factors promote IPF remain unclear. We aimed to identify genetic variants associated with IPF susceptibility and provide mechanistic insight using gene and protein expression analyses.

602 European ancestry cases, 3,366 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

11321
Total Participants
GWAS
Study Type
Yes
Replicated
2,158 cases, 5,195 controls
Replication Participants
European
Ancestry
U.K., U.S.
Recruitment Country
Chapter IV

AI-Generated Summary

AI-generated by DNAGENICS

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