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GWAS Study

Genome-wide association study identifies susceptibility loci for B-cell childhood acute lymphoblastic leukemia.

Vijayakrishnan J, Studd J, Broderick P et al.

29632299 PubMed ID
GWAS Study Type
17051 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

VJ
Vijayakrishnan J
SJ
Studd J
BP
Broderick P
KB
Kinnersley B
HA
Holroyd A
LP
Law PJ
KR
Kumar R
AJ
Allan JM
HC
Harrison CJ
MA
Moorman AV
VA
Vora A
RE
Roman E
RS
Rachakonda S
KS
Kinsey SE
SE
Sheridan E
TP
Thompson PD
IJ
Irving JA
KR
Koehler R
HP
Hoffmann P
NM
Nöthen MM
HS
Heilmann-Heimbach S
JK
Jöckel KH
ED
Easton DF
PP
Pharaoh PDP
DA
Dunning AM
PJ
Peto J
CF
Canzian F
SA
Swerdlow A
ER
Eeles RA
KZ
Kote-Jarai Z
MK
Muir K
PN
Pashayan N
GM
Greaves M
ZM
Zimmerman M
BC
Bartram CR
SM
Schrappe M
SM
Stanulla M
HK
Hemminki K
HR
Houlston RS
Chapter II

Abstract

Summary of the research findings

Genome-wide association studies (GWAS) have advanced our understanding of susceptibility to B-cell precursor acute lymphoblastic leukemia (BCP-ALL); however, much of the heritable risk remains unidentified. Here, we perform a GWAS and conduct a meta-analysis with two existing GWAS, totaling 2442 cases and 14,609 controls. We identify risk loci for BCP-ALL at 8q24.21 (rs28665337, P = 3.86 × 10-9, odds ratio (OR) = 1.34) and for ETV6-RUNX1 fusion-positive BCP-ALL at 2q22.3 (rs17481869, P = 3.20 × 10-8, OR = 2.14). Our findings provide further insights into genetic susceptibility to ALL and its biology.

2,442 European ancestry cases, 14,609 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

17051
Total Participants
GWAS
Study Type
No
Replicated
European
Ancestry
Germany, U.K.
Recruitment Country
Chapter IV

AI-Generated Summary

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