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GWAS Study

Genetic Regulation of Atherosclerosis-Relevant Phenotypes in Human Vascular Smooth Muscle Cells.

Aherrahrou R, Guo L, Nagraj VP et al.

33040646 PubMed ID
GWAS Study Type
151 Participants
156 Views
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

AR
Aherrahrou R
GL
Guo L
NV
Nagraj VP
AA
Aguhob A
HJ
Hinkle J
CL
Chen L
YS
Yuhl Soh J
LD
Lue D
AG
Alencar GF
BA
Boltjes A
VD
van der Laan SW
FE
Farber E
FD
Fuller D
AR
Anane-Wae R
AN
Akingbesote N
MA
Manichaikul AW
ML
Ma L
KM
Kaikkonen MU
BJ
Björkegren JLM
ÖS
Önengüt-Gümüşcü S
PG
Pasterkamp G
MC
Miller CL
OG
Owens GK
FA
Finn A
NM
Navab M
FA
Fogelman AM
BJ
Berliner JA
CM
Civelek M
Chapter II

Abstract

Summary of the research findings

Rationale: Coronary artery disease (CAD) is a major cause of morbidity and mortality worldwide. Recent genome-wide association studies revealed 163 loci associated with CAD. However, the precise molecular mechanisms by which the majority of these loci increase CAD risk are not known. Vascular smooth muscle cells (VSMCs) are critical in the development of CAD. They can play either beneficial or detrimental roles in lesion pathogenesis, depending on the nature of their phenotypic changes.

69 European ancestry individuals, 64 Hispanic/Latin American individuals, 12 African ancestry individuals, 6 East Asian ancestry individuals

Chapter III

Study Statistics

Key metrics and study information

151
Total Participants
GWAS
Study Type
No
Replicated
European, African unspecified, East Asian, Hispanic or Latin American
Ancestry
Chapter IV

AI-Generated Summary

AI-generated by DNAGENICS

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