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GWAS Study

Phenome-wide analysis of Taiwan Biobank reveals novel glycemia-related loci and genetic risks for diabetes.

Lee CJ, Chen TH, Lim AMW et al.

36329257 PubMed ID
GWAS Study Type
64429 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

LC
Lee CJ
CT
Chen TH
LA
Lim AMW
CC
Chang CC
SJ
Sie JJ
CP
Chen PL
CS
Chang SW
WS
Wu SJ
HC
Hsu CL
HA
Hsieh AR
YW
Yang WS
FC
Fann CSJ
Chapter II

Abstract

Summary of the research findings

To explore the complex genetic architecture of common diseases and traits, we conducted comprehensive PheWAS of ten diseases and 34 quantitative traits in the community-based Taiwan Biobank (TWB). We identified 995 significantly associated loci with 135 novel loci specific to Taiwanese population. Further analyses highlighted the genetic pleiotropy of loci related to complex disease and associated quantitative traits. Extensive analysis on glycaemic phenotypes (T2D, fasting glucose and HbA1c) was performed and identified 115 significant loci with four novel genetic variants (HACL1, RAD21, ASH1L and GAK). Transcriptomics data also strengthen the relevancy of the findings to metabolic disorders, thus contributing to better understanding of pathogenesis. In addition, genetic risk scores are constructed and validated for absolute risks prediction of T2D in Taiwanese population. In conclusion, our data-driven approach without a priori hypothesis is useful for novel gene discovery and validation on top of disease risk prediction for unique non-European population.

5,096 Taiwanese ancestry cases, 59,333 Taiwanese ancestry controls

Chapter III

Study Statistics

Key metrics and study information

64429
Total Participants
GWAS
Study Type
No
Replicated
East Asian
Ancestry
Taiwan
Recruitment Country
Chapter IV

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