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GWAS Study

Large-scale cross-ancestry genome-wide meta-analysis of serum urate.

Cho C, Kim B, Kim DS et al.

38658550 PubMed ID
GWAS Study Type
1029323 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

CC
Cho C
KB
Kim B
KD
Kim DS
HM
Hwang MY
SI
Shim I
SM
Song M
LY
Lee YC
JS
Jung SH
CS
Cho SK
PW
Park WY
MW
Myung W
KB
Kim BJ
DR
Do R
CH
Choi HK
MT
Merriman TR
KY
Kim YJ
WH
Won HH
Chapter II

Abstract

Summary of the research findings

Hyperuricemia is an essential causal risk factor for gout and is associated with cardiometabolic diseases. Given the limited contribution of East Asian ancestry to genome-wide association studies of serum urate, the genetic architecture of serum urate requires exploration. A large-scale cross-ancestry genome-wide association meta-analysis of 1,029,323 individuals and ancestry-specific meta-analysis identifies a total of 351 loci, including 17 previously unreported loci. The genetic architecture of serum urate control is similar between European and East Asian populations. A transcriptome-wide association study, enrichment analysis, and colocalization analysis in relevant tissues identify candidate serum urate-associated genes, including CTBP1, SKIV2L, and WWP2. A phenome-wide association study using polygenic risk scores identifies serum urate-correlated diseases including heart failure and hypertension. Mendelian randomization and mediation analyses show that serum urate-associated genes might have a causal relationship with serum urate-correlated diseases via mediation effects. This study elucidates our understanding of the genetic architecture of serum urate control.

219,768 East Asian ancestry individuals, 677,373 European ancestry individuals, 132,182 individuals

Chapter III

Study Statistics

Key metrics and study information

1029323
Total Participants
GWAS
Study Type
No
Replicated
East Asian, European
Ancestry
Japan, Republic of Korea, U.S., U.K.
Recruitment Country
Chapter IV

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