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GWAS Study

Genome-wide association analyses identify 44 risk variants and refine the genetic architecture of major depression.

Wray NR, Ripke S, Mattheisen M et al.

29700475 PubMed ID
GWAS Study Type
480359 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

WN
Wray NR
RS
Ripke S
MM
Mattheisen M
TM
Trzaskowski M
BE
Byrne EM
AA
Abdellaoui A
AM
Adams MJ
AE
Agerbo E
AT
Air TM
AT
Andlauer TMF
BS
Bacanu SA
BM
Bækvad-Hansen M
BA
Beekman AFT
BT
Bigdeli TB
BE
Binder EB
BD
Blackwood DRH
BJ
Bryois J
BH
Buttenschøn HN
BJ
Bybjerg-Grauholm J
CN
Cai N
CE
Castelao E
CJ
Christensen JH
CT
Clarke TK
CJ
Coleman JIR
CL
Colodro-Conde L
CB
Couvy-Duchesne B
CN
Craddock N
CG
Crawford GE
CC
Crowley CA
DH
Dashti HS
DG
Davies G
DI
Deary IJ
DF
Degenhardt F
DE
Derks EM
DN
Direk N
DC
Dolan CV
DE
Dunn EC
ET
Eley TC
EN
Eriksson N
EV
Escott-Price V
KF
Kiadeh FHF
FH
Finucane HK
FA
Forstner AJ
FJ
Frank J
GH
Gaspar HA
GM
Gill M
GP
Giusti-Rodríguez P
GF
Goes FS
GS
Gordon SD
GJ
Grove J
HL
Hall LS
HE
Hannon E
HC
Hansen CS
HT
Hansen TF
HS
Herms S
HI
Hickie IB
HP
Hoffmann P
HG
Homuth G
HC
Horn C
HJ
Hottenga JJ
HD
Hougaard DM
HM
Hu M
HC
Hyde CL
IM
Ising M
JR
Jansen R
JF
Jin F
JE
Jorgenson E
KJ
Knowles JA
KI
Kohane IS
KJ
Kraft J
KW
Kretzschmar WW
KJ
Krogh J
KZ
Kutalik Z
LJ
Lane JM
LY
Li Y
LY
Li Y
LP
Lind PA
LX
Liu X
LL
Lu L
MD
MacIntyre DJ
MD
MacKinnon DF
MR
Maier RM
MW
Maier W
MJ
Marchini J
MH
Mbarek H
MP
McGrath P
MP
McGuffin P
MS
Medland SE
MD
Mehta D
MC
Middeldorp CM
ME
Mihailov E
MY
Milaneschi Y
ML
Milani L
MJ
Mill J
MF
Mondimore FM
MG
Montgomery GW
MS
Mostafavi S
MN
Mullins N
NM
Nauck M
NB
Ng B
NM
Nivard MG
ND
Nyholt DR
OP
O'Reilly PF
OH
Oskarsson H
OM
Owen MJ
PJ
Painter JN
PC
Pedersen CB
PM
Pedersen MG
PR
Peterson RE
PE
Pettersson E
PW
Peyrot WJ
PG
Pistis G
PD
Posthuma D
PS
Purcell SM
QJ
Quiroz JA
QP
Qvist P
RJ
Rice JP
RB
Riley BP
RM
Rivera M
SM
Saeed Mirza S
SR
Saxena R
SR
Schoevers R
SE
Schulte EC
SL
Shen L
SJ
Shi J
SS
Shyn SI
SE
Sigurdsson E
SG
Sinnamon GBC
SJ
Smit JH
SD
Smith DJ
SH
Stefansson H
SS
Steinberg S
SC
Stockmeier CA
SF
Streit F
SJ
Strohmaier J
TK
Tansey KE
TH
Teismann H
TA
Teumer A
TW
Thompson W
TP
Thomson PA
TT
Thorgeirsson TE
TC
Tian C
TM
Traylor M
TJ
Treutlein J
TV
Trubetskoy V
UA
Uitterlinden AG
UD
Umbricht D
VD
Van der Auwera S
VH
van Hemert AM
VA
Viktorin A
VP
Visscher PM
WY
Wang Y
WB
Webb BT
WS
Weinsheimer SM
WJ
Wellmann J
WG
Willemsen G
WS
Witt SH
WY
Wu Y
XH
Xi HS
YJ
Yang J
ZF
Zhang F
AV
Arolt V
BB
Baune BT
BK
Berger K
BD
Boomsma DI
CS
Cichon S
DU
Dannlowski U
DG
de Geus ECJ
DJ
DePaulo JR
DE
Domenici E
DK
Domschke K
ET
Esko T
GH
Grabe HJ
HS
Hamilton SP
HC
Hayward C
HA
Heath AC
HD
Hinds DA
KK
Kendler KS
KS
Kloiber S
LG
Lewis G
LQ
Li QS
LS
Lucae S
MP
Madden PFA
MP
Magnusson PK
MN
Martin NG
MA
McIntosh AM
MA
Metspalu A
MO
Mors O
MP
Mortensen PB
MB
Müller-Myhsok B
NM
Nordentoft M
NM
Nöthen MM
OM
O'Donovan MC
PS
Paciga SA
PN
Pedersen NL
PB
Penninx BWJH
PR
Perlis RH
PD
Porteous DJ
PJ
Potash JB
PM
Preisig M
RM
Rietschel M
SC
Schaefer C
ST
Schulze TG
SJ
Smoller JW
SK
Stefansson K
TH
Tiemeier H
UR
Uher R
VH
Völzke H
WM
Weissman MM
WT
Werge T
WA
Winslow AR
LC
Lewis CM
LD
Levinson DF
BG
Breen G
BA
Børglum AD
SP
Sullivan PF
Chapter II

Abstract

Summary of the research findings

Major depressive disorder (MDD) is a common illness accompanied by considerable morbidity, mortality, costs, and heightened risk of suicide. We conducted a genome-wide association meta-analysis based in 135,458 cases and 344,901 controls and identified 44 independent and significant loci. The genetic findings were associated with clinical features of major depression and implicated brain regions exhibiting anatomical differences in cases. Targets of antidepressant medications and genes involved in gene splicing were enriched for smaller association signal. We found important relationships of genetic risk for major depression with educational attainment, body mass, and schizophrenia: lower educational attainment and higher body mass were putatively causal, whereas major depression and schizophrenia reflected a partly shared biological etiology. All humans carry lesser or greater numbers of genetic risk factors for major depression. These findings help refine the basis of major depression and imply that a continuous measure of risk underlies the clinical phenotype.

135,458 European ancestry cases, 344,901 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

480359
Total Participants
GWAS
Study Type
No
Replicated
European
Ancestry
U.S., Australia, Germany, Netherlands, Switzerland, Denmark, Iceland, Republic of Ireland, Sweden, U.K.
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

Important Disclaimer: This review has been performed semi-automatically and is provided for informational purposes only. While we strive for accuracy, this analysis may contain errors, omissions, or misinterpretations of the original research. DNA Genics disclaims all liability for any inaccuracies, errors, or consequences arising from the use of this information. Users should independently verify all information and consult original research publications before making any decisions based on this content. This analysis is not intended as a substitute for professional scientific review or medical advice.

Analysis In Progress

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